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Times Cited: 350
Times Cited: 350
Times Cited
Times Co-cited
Similarity
Genome-wide association analyses identify 44 risk variants and refine the genetic architecture of major depression.
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Genome-wide meta-analysis of depression identifies 102 independent variants and highlights the importance of the prefrontal brain regions.
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Genome-wide association study of depression phenotypes in UK Biobank identifies variants in excitatory synaptic pathways.
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Genetic variants associated with subjective well-being, depressive symptoms, and neuroticism identified through genome-wide analyses.
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Functional mapping and annotation of genetic associations with FUMA.
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A mega-analysis of genome-wide association studies for major depressive disorder.
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An atlas of genetic correlations across human diseases and traits.
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MAGMA: generalized gene-set analysis of GWAS data.
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Gene discovery and polygenic prediction from a genome-wide association study of educational attainment in 1.1 million individuals.
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Minimal phenotyping yields genome-wide association signals of low specificity for major depression.
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PLINK: a tool set for whole-genome association and population-based linkage analyses.
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Genetic relationship between five psychiatric disorders estimated from genome-wide SNPs.
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A global reference for human genetic variation.
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Genome-wide association study identifies 30 loci associated with bipolar disorder.
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Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection.
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Meta-analysis of genome-wide association studies for neuroticism in 449,484 individuals identifies novel genetic loci and pathways.
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Genome-wide association meta-analysis in 269,867 individuals identifies new genetic and functional links to intelligence.
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METAL: fast and efficient meta-analysis of genomewide association scans.
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Genome-wide polygenic scores for common diseases identify individuals with risk equivalent to monogenic mutations.
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An Analysis of Two Genome-wide Association Meta-analyses Identifies a New Locus for Broad Depression Phenotype.
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Discovery of the first genome-wide significant risk loci for attention deficit/hyperactivity disorder.
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Uncovering the Genetic Architecture of Major Depression.
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UK biobank: an open access resource for identifying the causes of a wide range of complex diseases of middle and old age.
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Negr1 controls adult hippocampal neurogenesis and affective behaviors.
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Genomic structural equation modelling provides insights into the multivariate genetic architecture of complex traits.
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Causal associations between risk factors and common diseases inferred from GWAS summary data.
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Largest GWAS of PTSD (N=20 070) yields genetic overlap with schizophrenia and sex differences in heritability.
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No Support for Historical Candidate Gene or Candidate Gene-by-Interaction Hypotheses for Major Depression Across Multiple Large Samples.
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Study of 300,486 individuals identifies 148 independent genetic loci influencing general cognitive function.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.