Rahul Bhowmick, Sheroy Minocherhomji, Ian D Hickson. Mol Cell 2016
Times Cited: 191
Times Cited: 191
Times Cited
Times Co-cited
Similarity
Replication stress activates DNA repair synthesis in mitosis.
Sheroy Minocherhomji, Songmin Ying, Victoria A Bjerregaard, Sara Bursomanno, Aiste Aleliunaite, Wei Wu, Hocine W Mankouri, Huahao Shen, Ying Liu, Ian D Hickson. Nature 2015
Sheroy Minocherhomji, Songmin Ying, Victoria A Bjerregaard, Sara Bursomanno, Aiste Aleliunaite, Wei Wu, Hocine W Mankouri, Huahao Shen, Ying Liu, Ian D Hickson. Nature 2015
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Mammalian RAD52 Functions in Break-Induced Replication Repair of Collapsed DNA Replication Forks.
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Break-induced replication repair of damaged forks induces genomic duplications in human cells.
Lorenzo Costantino, Sotirios K Sotiriou, Juha K Rantala, Simon Magin, Emil Mladenov, Thomas Helleday, James E Haber, George Iliakis, Olli P Kallioniemi, Thanos D Halazonetis. Science 2014
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Alternative Lengthening of Telomeres Mediated by Mitotic DNA Synthesis Engages Break-Induced Replication Processes.
Jaewon Min, Woodring E Wright, Jerry W Shay. Mol Cell Biol 2017
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Break-induced telomere synthesis underlies alternative telomere maintenance.
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Double-strand break repair-independent role for BRCA2 in blocking stalled replication fork degradation by MRE11.
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Causes and consequences of replication stress.
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MUS81 promotes common fragile site expression.
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Replication stress induces sister-chromatid bridging at fragile site loci in mitosis.
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Alternative Lengthening of Telomeres through Two Distinct Break-Induced Replication Pathways.
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Rad52 inactivation is synthetically lethal with BRCA2 deficiency.
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ERCC1 and MUS81-EME1 promote sister chromatid separation by processing late replication intermediates at common fragile sites during mitosis.
Valeria Naim, Therese Wilhelm, Michelle Debatisse, Filippo Rosselli. Nat Cell Biol 2013
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RAD52 and SLX4 act nonepistatically to ensure telomere stability during alternative telomere lengthening.
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Break-induced replication and telomerase-independent telomere maintenance require Pol32.
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DNA REPAIR. Mus81 and converging forks limit the mutagenicity of replication fork breakage.
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Rad51-mediated replication fork reversal is a global response to genotoxic treatments in human cells.
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Alternative lengthening of human telomeres is a conservative DNA replication process with features of break-induced replication.
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A distinct replication fork protection pathway connects Fanconi anemia tumor suppressors to RAD51-BRCA1/2.
Katharina Schlacher, Hong Wu, Maria Jasin. Cancer Cell 2012
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53BP1 nuclear bodies form around DNA lesions generated by mitotic transmission of chromosomes under replication stress.
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RAD52 inactivation is synthetically lethal with deficiencies in BRCA1 and PALB2 in addition to BRCA2 through RAD51-mediated homologous recombination.
B H Lok, A C Carley, B Tchang, S N Powell. Oncogene 2013
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Human cancer cells utilize mitotic DNA synthesis to resist replication stress at telomeres regardless of their telomere maintenance mechanism.
Özgün Özer, Rahul Bhowmick, Ying Liu, Ian D Hickson. Oncotarget 2018
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Migrating bubble during break-induced replication drives conservative DNA synthesis.
Natalie Saini, Sreejith Ramakrishnan, Rajula Elango, Sandeep Ayyar, Yu Zhang, Angela Deem, Grzegorz Ira, James E Haber, Kirill S Lobachev, Anna Malkova. Nature 2013
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Replication fork reversal triggers fork degradation in BRCA2-defective cells.
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Intragenic origins due to short G1 phases underlie oncogene-induced DNA replication stress.
Morgane Macheret, Thanos D Halazonetis. Nature 2018
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Fragile sites in cancer: more than meets the eye.
Thomas W Glover, Thomas E Wilson, Martin F Arlt. Nat Rev Cancer 2017
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Unresolved recombination intermediates lead to ultra-fine anaphase bridges, chromosome breaks and aberrations.
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DNA synthesis by Pol η promotes fragile site stability by preventing under-replicated DNA in mitosis.
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Replication Fork Slowing and Reversal upon DNA Damage Require PCNA Polyubiquitination and ZRANB3 DNA Translocase Activity.
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A Mechanism for Controlled Breakage of Under-replicated Chromosomes during Mitosis.
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Collisions between replication and transcription complexes cause common fragile site instability at the longest human genes.
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Break-induced replication occurs by conservative DNA synthesis.
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The essential kinase ATR: ensuring faithful duplication of a challenging genome.
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RECQ5 Helicase Cooperates with MUS81 Endonuclease in Processing Stalled Replication Forks at Common Fragile Sites during Mitosis.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.