Nicolas Snaidero, Mikael Simons. Glia 2017
Times Cited: 48
Times Cited: 48
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Actin filament turnover drives leading edge growth during myelin sheath formation in the central nervous system.
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Antagonistic Functions of MBP and CNP Establish Cytosolic Channels in CNS Myelin.
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Glycolytic oligodendrocytes maintain myelin and long-term axonal integrity.
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Phosphatidylinositol 4,5-bisphosphate-dependent interaction of myelin basic protein with the plasma membrane in oligodendroglial cells and its rapid perturbation by elevated calcium.
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Oligodendrocyte heterogeneity in the mouse juvenile and adult central nervous system.
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Neuronal activity promotes oligodendrogenesis and adaptive myelination in the mammalian brain.
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Somatodendritic Expression of JAM2 Inhibits Oligodendrocyte Myelination.
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Myelin membrane assembly is driven by a phase transition of myelin basic proteins into a cohesive protein meshwork.
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Individual oligodendrocytes have only a few hours in which to generate new myelin sheaths in vivo.
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Activation of the mammalian target of rapamycin (mTOR) is essential for oligodendrocyte differentiation.
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HDAC1 and HDAC2 regulate oligodendrocyte differentiation by disrupting the beta-catenin-TCF interaction.
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ERK1/ERK2 MAPK signaling is required to increase myelin thickness independent of oligodendrocyte differentiation and initiation of myelination.
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From axon-glial signalling to myelination: the integrating role of oligodendroglial Fyn kinase.
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