Samah W Awwad, Enas R Abu-Zhayia, Noga Guttmann-Raviv, Nabieh Ayoub. EMBO Rep 2017
Times Cited: 43
Times Cited: 43
Times Cited
Times Co-cited
Similarity
ATM-dependent chromatin changes silence transcription in cis to DNA double-strand breaks.
Niraj M Shanbhag, Ilona U Rafalska-Metcalf, Carlo Balane-Bolivar, Susan M Janicki, Roger A Greenberg. Cell 2010
Niraj M Shanbhag, Ilona U Rafalska-Metcalf, Carlo Balane-Bolivar, Susan M Janicki, Roger A Greenberg. Cell 2010
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DNAPKcs-dependent arrest of RNA polymerase II transcription in the presence of DNA breaks.
Tibor Pankotai, Céline Bonhomme, David Chen, Evi Soutoglou. Nat Struct Mol Biol 2012
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Requirement for PBAF in transcriptional repression and repair at DNA breaks in actively transcribed regions of chromatin.
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Transcriptional elongation factor ENL phosphorylated by ATM recruits polycomb and switches off transcription for DSB repair.
Ayako Ui, Yuko Nagaura, Akira Yasui. Mol Cell 2015
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Senataxin resolves RNA:DNA hybrids forming at DNA double-strand breaks to prevent translocations.
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A chromatin localization screen reveals poly (ADP ribose)-regulated recruitment of the repressive polycomb and NuRD complexes to sites of DNA damage.
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Screen identifies bromodomain protein ZMYND8 in chromatin recognition of transcription-associated DNA damage that promotes homologous recombination.
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WWP2 ubiquitylates RNA polymerase II for DNA-PK-dependent transcription arrest and repair at DNA breaks.
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Transcriptionally active chromatin recruits homologous recombination at DNA double-strand breaks.
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Histone demethylase KDM5A regulates the ZMYND8-NuRD chromatin remodeler to promote DNA repair.
Fade Gong, Thomas Clouaire, Marion Aguirrebengoa, Gaëlle Legube, Kyle M Miller. J Cell Biol 2017
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Damage-induced lncRNAs control the DNA damage response through interaction with DDRNAs at individual double-strand breaks.
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A damaged genome's transcriptional landscape through multilayered expression profiling around in situ-mapped DNA double-strand breaks.
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Human Rad52 Promotes XPG-Mediated R-loop Processing to Initiate Transcription-Associated Homologous Recombination Repair.
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Acetylation limits 53BP1 association with damaged chromatin to promote homologous recombination.
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CDYL1 fosters double-strand break-induced transcription silencing and promotes homology-directed repair.
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Repression of Transcription at DNA Breaks Requires Cohesin throughout Interphase and Prevents Genome Instability.
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BRCA2 controls DNA:RNA hybrid level at DSBs by mediating RNase H2 recruitment.
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The ATM repair pathway inhibits RNA polymerase I transcription in response to chromosome breaks.
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Transient RNA-DNA Hybrids Are Required for Efficient Double-Strand Break Repair.
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Drosha drives the formation of DNA:RNA hybrids around DNA break sites to facilitate DNA repair.
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Site-specific DICER and DROSHA RNA products control the DNA-damage response.
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Chemical genetic discovery of PARP targets reveals a role for PARP-1 in transcription elongation.
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DNA double-strand breaks promote methylation of histone H3 on lysine 9 and transient formation of repressive chromatin.
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RNF168 ubiquitinates K13-15 on H2A/H2AX to drive DNA damage signaling.
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Single-molecule imaging of transcription at damaged chromatin.
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NELF, a multisubunit complex containing RD, cooperates with DSIF to repress RNA polymerase II elongation.
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BMI1-UBR5 axis regulates transcriptional repression at damaged chromatin.
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Transcription-Coupled DNA Double-Strand Break Repair: Active Genes Need Special Care.
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Tyrosine kinase c-Abl couples RNA polymerase II transcription to DNA double-strand breaks.
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Transcription and DNA Damage: Holding Hands or Crossing Swords?
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BMI1 is recruited to DNA breaks and contributes to DNA damage-induced H2A ubiquitination and repair.
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Comprehensive Mapping of Histone Modifications at DNA Double-Strand Breaks Deciphers Repair Pathway Chromatin Signatures.
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Bon voyage: A transcriptional journey around DNA breaks.
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Histone H1 couples initiation and amplification of ubiquitin signalling after DNA damage.
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Polycomb repressive complex 2 contributes to DNA double-strand break repair.
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The DNA damage response: making it safe to play with knives.
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Genome-wide mapping of long-range contacts unveils clustering of DNA double-strand breaks at damaged active genes.
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PARP1-dependent recruitment of the FBXL10-RNF68-RNF2 ubiquitin ligase to sites of DNA damage controls H2A.Z loading.
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DEAD Box 1 Facilitates Removal of RNA and Homologous Recombination at DNA Double-Strand Breaks.
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ATM and ATR substrate analysis reveals extensive protein networks responsive to DNA damage.
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Quantitative Proteomic Atlas of Ubiquitination and Acetylation in the DNA Damage Response.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.