Sara Ballouz, Jesse Gillis. Genome Med 2017
Times Cited: 8
Times Cited: 8
Times Cited
Times Co-cited
Similarity
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De novo mutations in schizophrenia implicate synaptic networks.
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Polygenic transmission disequilibrium confirms that common and rare variation act additively to create risk for autism spectrum disorders.
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Transcriptional signatures of schizophrenia in hiPSC-derived NPCs and neurons are concordant with post-mortem adult brains.
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A polygenic burden of rare disruptive mutations in schizophrenia.
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Contribution of copy number variants to schizophrenia from a genome-wide study of 41,321 subjects.
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Gene expression elucidates functional impact of polygenic risk for schizophrenia.
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An Expanded View of Complex Traits: From Polygenic to Omnigenic.
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Psychiatric genome-wide association study analyses implicate neuronal, immune and histone pathways.
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Phenotypic differences in hiPSC NPCs derived from patients with schizophrenia.
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Common alleles contribute to schizophrenia in CNV carriers.
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Human Neuropsychiatric Disease Modeling using Conditional Deletion Reveals Synaptic Transmission Defects Caused by Heterozygous Mutations in NRXN1.
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Open Chromatin Profiling in hiPSC-Derived Neurons Prioritizes Functional Noncoding Psychiatric Risk Variants and Highlights Neurodevelopmental Loci.
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Genetic identification of brain cell types underlying schizophrenia.
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Evaluating Synthetic Activation and Repression of Neuropsychiatric-Related Genes in hiPSC-Derived NPCs, Neurons, and Astrocytes.
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Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection.
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A model for neural development and treatment of Rett syndrome using human induced pluripotent stem cells.
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variancePartition: interpreting drivers of variation in complex gene expression studies.
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Synaptic, transcriptional and chromatin genes disrupted in autism.
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MicroRNA Profiling of Neurons Generated Using Induced Pluripotent Stem Cells Derived from Patients with Schizophrenia and Schizoaffective Disorder, and 22q11.2 Del.
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Sequencing chromosomal abnormalities reveals neurodevelopmental loci that confer risk across diagnostic boundaries.
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MAGMA: generalized gene-set analysis of GWAS data.
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Perturb-Seq: Dissecting Molecular Circuits with Scalable Single-Cell RNA Profiling of Pooled Genetic Screens.
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Human iPSC neurons display activity-dependent neurotransmitter secretion: aberrant catecholamine levels in schizophrenia neurons.
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Genomic DISC1 Disruption in hiPSCs Alters Wnt Signaling and Neural Cell Fate.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.