Michael W Salter, Beth Stevens. Nat Med 2017
Times Cited: 695
Times Cited: 695
Times Cited
Times Co-cited
Similarity
Fate mapping analysis reveals that adult microglia derive from primitive macrophages.
Florent Ginhoux, Melanie Greter, Marylene Leboeuf, Sayan Nandi, Peter See, Solen Gokhan, Mark F Mehler, Simon J Conway, Lai Guan Ng, E Richard Stanley,[...]. Science 2010
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Microglia sculpt postnatal neural circuits in an activity and complement-dependent manner.
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Neurotoxic reactive astrocytes are induced by activated microglia.
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A Unique Microglia Type Associated with Restricting Development of Alzheimer's Disease.
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Synaptic pruning by microglia is necessary for normal brain development.
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Identification of a unique TGF-β-dependent molecular and functional signature in microglia.
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Resting microglial cells are highly dynamic surveillants of brain parenchyma in vivo.
Axel Nimmerjahn, Frank Kirchhoff, Fritjof Helmchen. Science 2005
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An environment-dependent transcriptional network specifies human microglia identity.
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Microglia in Physiology and Disease.
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Single-Cell RNA Sequencing of Microglia throughout the Mouse Lifespan and in the Injured Brain Reveals Complex Cell-State Changes.
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Single-cell transcriptomic analysis of Alzheimer's disease.
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iPSC-Derived Human Microglia-like Cells to Study Neurological Diseases.
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Microglia Biology: One Century of Evolving Concepts.
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The TREM2-APOE Pathway Drives the Transcriptional Phenotype of Dysfunctional Microglia in Neurodegenerative Diseases.
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Microglia development follows a stepwise program to regulate brain homeostasis.
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Microglia promote learning-dependent synapse formation through brain-derived neurotrophic factor.
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Complement and microglia mediate early synapse loss in Alzheimer mouse models.
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Colony-stimulating factor 1 receptor signaling is necessary for microglia viability, unmasking a microglia progenitor cell in the adult brain.
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Microglia and macrophages in brain homeostasis and disease.
Qingyun Li, Ben A Barres. Nat Rev Immunol 2018
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Efficient derivation of microglia-like cells from human pluripotent stem cells.
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The microglial sensome revealed by direct RNA sequencing.
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New tools for studying microglia in the mouse and human CNS.
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A Highly Efficient Human Pluripotent Stem Cell Microglia Model Displays a Neuronal-Co-culture-Specific Expression Profile and Inflammatory Response.
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Differentiation of human and murine induced pluripotent stem cells to microglia-like cells.
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Microglia emerge from erythromyeloid precursors via Pu.1- and Irf8-dependent pathways.
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Microglia Function in the Central Nervous System During Health and Neurodegeneration.
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Innate immune memory in the brain shapes neurological disease hallmarks.
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Cross-Species Single-Cell Analysis Reveals Divergence of the Primate Microglia Program.
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ATP mediates rapid microglial response to local brain injury in vivo.
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Developmental Heterogeneity of Microglia and Brain Myeloid Cells Revealed by Deep Single-Cell RNA Sequencing.
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Astrocyte-Microglia Cross Talk through Complement Activation Modulates Amyloid Pathology in Mouse Models of Alzheimer's Disease.
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Inflammasome signalling in brain function and neurodegenerative disease.
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Tissue-resident macrophages originate from yolk-sac-derived erythro-myeloid progenitors.
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Microglia regulate the number of neural precursor cells in the developing cerebral cortex.
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Reactive astrocyte nomenclature, definitions, and future directions.
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Physiology of microglia.
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Transcriptomic analysis of purified human cortical microglia reveals age-associated changes.
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The Microglial Innate Immune Receptor TREM2 Is Required for Synapse Elimination and Normal Brain Connectivity.
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A 3D human triculture system modeling neurodegeneration and neuroinflammation in Alzheimer's disease.
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Microglial brain region-dependent diversity and selective regional sensitivities to aging.
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Microglia: new roles for the synaptic stripper.
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Human and mouse single-nucleus transcriptomics reveal TREM2-dependent and TREM2-independent cellular responses in Alzheimer's disease.
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Microglial control of astrocytes in response to microbial metabolites.
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Lipid-droplet-accumulating microglia represent a dysfunctional and proinflammatory state in the aging brain.
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Microglia turnover with aging and in an Alzheimer's model via long-term in vivo single-cell imaging.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.