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Times Cited: 91
Times Cited
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Similarity
HRDetect is a predictor of BRCA1 and BRCA2 deficiency based on mutational signatures.
Helen Davies, Dominik Glodzik, Sandro Morganella, Lucy R Yates, Johan Staaf, Xueqing Zou, Manasa Ramakrishna, Sancha Martin, Sandrine Boyault, Anieta M Sieuwerts,[...]. Nat Med 2017
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Homologous Recombination Deficiency (HRD) Score Predicts Response to Platinum-Containing Neoadjuvant Chemotherapy in Patients with Triple-Negative Breast Cancer.
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Landscape of somatic mutations in 560 breast cancer whole-genome sequences.
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The repertoire of mutational signatures in human cancer.
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Patterns of genomic loss of heterozygosity predict homologous recombination repair defects in epithelial ovarian cancer.
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Telomeric allelic imbalance indicates defective DNA repair and sensitivity to DNA-damaging agents.
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Association of BRCA1/2 defects with genomic scores predictive of DNA damage repair deficiency among breast cancer subtypes.
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Mutational processes molding the genomes of 21 breast cancers.
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DeconstructSigs: delineating mutational processes in single tumors distinguishes DNA repair deficiencies and patterns of carcinoma evolution.
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Olaparib plus Bevacizumab as First-Line Maintenance in Ovarian Cancer.
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Homologous recombination DNA repair deficiency and PARP inhibition activity in primary triple negative breast cancer.
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The Sequence Alignment/Map format and SAMtools.
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Germline Mutation Status, Pathological Complete Response, and Disease-Free Survival in Triple-Negative Breast Cancer: Secondary Analysis of the GeparSixto Randomized Clinical Trial.
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Rucaparib in relapsed, platinum-sensitive high-grade ovarian carcinoma (ARIEL2 Part 1): an international, multicentre, open-label, phase 2 trial.
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Migrating the SNP array-based homologous recombination deficiency measures to next generation sequencing data of breast cancer.
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Homologous recombination deficiency (HRD) status predicts response to standard neoadjuvant chemotherapy in patients with triple-negative or BRCA1/2 mutation-associated breast cancer.
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A RAD51 assay feasible in routine tumor samples calls PARP inhibitor response beyond BRCA mutation.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.