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Times Cited: 29
Times Cited: 29
Times Cited
Times Co-cited
Similarity
Mutations driving CLL and their evolution in progression and relapse.
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Complex karyotypes and KRAS and POT1 mutations impact outcome in CLL after chlorambucil-based chemotherapy or chemoimmunotherapy.
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Unmutated Ig V(H) genes are associated with a more aggressive form of chronic lymphocytic leukemia.
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Gene mutations and treatment outcome in chronic lymphocytic leukemia: results from the CLL8 trial.
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Extended follow-up and impact of high-risk prognostic factors from the phase 3 RESONATE study in patients with previously treated CLL/SLL.
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Complex karyotype is a stronger predictor than del(17p) for an inferior outcome in relapsed or refractory chronic lymphocytic leukemia patients treated with ibrutinib-based regimens.
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Integrated mutational and cytogenetic analysis identifies new prognostic subgroups in chronic lymphocytic leukemia.
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Single-agent ibrutinib in treatment-naïve and relapsed/refractory chronic lymphocytic leukemia: a 5-year experience.
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Clonal diversity predicts adverse outcome in chronic lymphocytic leukemia.
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Genetic and Functional Drivers of Diffuse Large B Cell Lymphoma.
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Burkitt lymphoma pathogenesis and therapeutic targets from structural and functional genomics.
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Fludarabine, cyclophosphamide, and rituximab chemoimmunotherapy is highly effective treatment for relapsed patients with CLL.
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First-line chemoimmunotherapy with bendamustine and rituximab versus fludarabine, cyclophosphamide, and rituximab in patients with advanced chronic lymphocytic leukaemia (CLL10): an international, open-label, randomised, phase 3, non-inferiority trial.
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Venetoclax-Rituximab in Relapsed or Refractory Chronic Lymphocytic Leukemia.
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Targeting BCL2 with Venetoclax in Relapsed Chronic Lymphocytic Leukemia.
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Long-term remissions after FCR chemoimmunotherapy in previously untreated patients with CLL: updated results of the CLL8 trial.
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Clinicopathological features and outcomes of progression of CLL on the BCL2 inhibitor venetoclax.
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Analysis of the chronic lymphocytic leukemia coding genome: role of NOTCH1 mutational activation.
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Mutations in the RAS-BRAF-MAPK-ERK pathway define a specific subgroup of patients with adverse clinical features and provide new therapeutic options in chronic lymphocytic leukemia.
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Transposon Mutagenesis Reveals Fludarabine Resistance Mechanisms in Chronic Lymphocytic Leukemia.
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Exome sequencing identifies recurrent mutations of the splicing factor SF3B1 gene in chronic lymphocytic leukemia.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.