A citation-based method for searching scientific literature

Samuel M Cohen, Benjamin Suutari, Xingzhi He, Yang Wang, Sandrine Sanchez, Natasha N Tirko, Nataniel J Mandelberg, Caitlin Mullins, Guangjun Zhou, Shuqi Wang, Ilona Kats, Alejandro Salah, Richard W Tsien, Huan Ma. Nat Commun 2018
Times Cited: 32







List of co-cited articles
392 articles co-cited >1



Times Cited
  Times     Co-cited
Similarity


γCaMKII shuttles Ca²⁺/CaM to the nucleus to trigger CREB phosphorylation and gene expression.
Huan Ma, Rachel D Groth, Samuel M Cohen, John F Emery, Boxing Li, Esthelle Hoedt, Guoan Zhang, Thomas A Neubert, Richard W Tsien. Cell 2014
163
65


The molecular basis of CaMKII function in synaptic and behavioural memory.
J Lisman, H Schulman, H Cline. Nat Rev Neurosci 2002
28

Mechanisms of CaMKII action in long-term potentiation.
John Lisman, Ryohei Yasuda, Sridhar Raghavachari. Nat Rev Neurosci 2012
660
25

Diagnostic exome sequencing in persons with severe intellectual disability.
Joep de Ligt, Marjolein H Willemsen, Bregje W M van Bon, Tjitske Kleefstra, Helger G Yntema, Thessa Kroes, Anneke T Vulto-van Silfhout, David A Koolen, Petra de Vries, Christian Gilissen,[...]. N Engl J Med 2012
25

Excitation-Transcription Coupling in Parvalbumin-Positive Interneurons Employs a Novel CaM Kinase-Dependent Pathway Distinct from Excitatory Neurons.
Samuel M Cohen, Huan Ma, Kishore V Kuchibhotla, Brendon O Watson, György Buzsáki, Robert C Froemke, Richard W Tsien. Neuron 2016
57
21

Identification of a genetic cluster influencing memory performance and hippocampal activity in humans.
Dominique J-F de Quervain, Andreas Papassotiropoulos. Proc Natl Acad Sci U S A 2006
116
18

Activation of CaMKII in single dendritic spines during long-term potentiation.
Seok-Jin R Lee, Yasmin Escobedo-Lozoya, Erzsebet M Szatmari, Ryohei Yasuda. Nature 2009
448
18

Autophosphorylation at Thr286 of the alpha calcium-calmodulin kinase II in LTP and learning.
K P Giese, N B Fedorov, R K Filipkowski, A J Silva. Science 1998
802
18


Ca(V)1 and Ca(V)2 channels engage distinct modes of Ca(2+) signaling to control CREB-dependent gene expression.
Damian G Wheeler, Rachel D Groth, Huan Ma, Curtis F Barrett, Scott F Owen, Parsa Safa, Richard W Tsien. Cell 2012
186
18

Impaired spatial learning in alpha-calcium-calmodulin kinase II mutant mice.
A J Silva, R Paylor, J M Wehner, S Tonegawa. Science 1992
15


βCaMKII plays a nonenzymatic role in hippocampal synaptic plasticity and learning by targeting αCaMKII to synapses.
Nils Z Borgesius, Geeske M van Woerden, Gabrielle H S Buitendijk, Nanda Keijzer, Dick Jaarsma, Casper C Hoogenraad, Ype Elgersma. J Neurosci 2011
85
15

Inhibitory autophosphorylation of CaMKII controls PSD association, plasticity, and learning.
Ype Elgersma, Nikolai B Fedorov, Sami Ikonen, Esther S Choi, Minetta Elgersma, Ofelia M Carvalho, Karl Peter Giese, Alcino J Silva. Neuron 2002
211
15


Synaptic tagging and long-term potentiation.
U Frey, R G Morris. Nature 1997
15


A Novel Human CAMK2A Mutation Disrupts Dendritic Morphology and Synaptic Transmission, and Causes ASD-Related Behaviors.
Jason R Stephenson, Xiaohan Wang, Tyler L Perfitt, Walker P Parrish, Brian C Shonesy, Christian R Marks, Douglas P Mortlock, Terunaga Nakagawa, James S Sutcliffe, Roger J Colbran. J Neurosci 2017
55
15

A novel mechanism for Ca2+/calmodulin-dependent protein kinase II targeting to L-type Ca2+ channels that initiates long-range signaling to the nucleus.
Xiaohan Wang, Christian R Marks, Tyler L Perfitt, Terunaga Nakagawa, Amy Lee, David A Jacobson, Roger J Colbran. J Biol Chem 2017
17
29

betaCaMKII controls the direction of plasticity at parallel fiber-Purkinje cell synapses.
Geeske M van Woerden, Freek E Hoebeek, Zhenyu Gao, Raghavendra Y Nagaraja, Casper C Hoogenraad, Steven A Kushner, Christian Hansel, Chris I De Zeeuw, Ype Elgersma. Nat Neurosci 2009
88
12


AlphaCaMKII autophosphorylation contributes to rapid learning but is not necessary for memory.
Elaine E Irvine, Jeffrey Vernon, K Peter Giese. Nat Neurosci 2005
99
12


Kinase-dead knock-in mouse reveals an essential role of kinase activity of Ca2+/calmodulin-dependent protein kinase IIalpha in dendritic spine enlargement, long-term potentiation, and learning.
Yoko Yamagata, Shizuka Kobayashi, Tatsuya Umeda, Akihiro Inoue, Hiroyuki Sakagami, Masahiro Fukaya, Masahiko Watanabe, Nobuhiko Hatanaka, Masako Totsuka, Takeshi Yagi,[...]. J Neurosci 2009
93
12

Long-distance integration of nuclear ERK signaling triggered by activation of a few dendritic spines.
Shenyu Zhai, Eugene D Ark, Paula Parra-Bueno, Ryohei Yasuda. Science 2013
75
12

Activity-dependent transport of the transcriptional coactivator CRTC1 from synapse to nucleus.
Toh Hean Ch'ng, Besim Uzgil, Peter Lin, Nuraly K Avliyakulov, Thomas J O'Dell, Kelsey C Martin. Cell 2012
135
12


Region-specific activation of CRTC1-CREB signaling mediates long-term fear memory.
Mio Nonaka, Ryang Kim, Hotaka Fukushima, Kazuki Sasaki, Kanzo Suzuki, Michiko Okamura, Yuichiro Ishii, Takashi Kawashima, Satoshi Kamijo, Sayaka Takemoto-Kimura,[...]. Neuron 2014
57
12


Learning induces long-term potentiation in the hippocampus.
Jonathan R Whitlock, Arnold J Heynen, Marshall G Shuler, Mark F Bear. Science 2006
12

Deficient long-term memory in mice with a targeted mutation of the cAMP-responsive element-binding protein.
R Bourtchuladze, B Frenguelli, J Blendy, D Cioffi, G Schutz, A J Silva. Cell 1994
12

A homozygous loss-of-function CAMK2A mutation causes growth delay, frequent seizures and severe intellectual disability.
Poh Hui Chia, Franklin Lei Zhong, Shinsuke Niwa, Carine Bonnard, Kagistia Hana Utami, Ruizhu Zeng, Hane Lee, Ascia Eskin, Stanley F Nelson, William H Xie,[...]. Elife 2018
31
12

The intellectual disability-associated CAMK2G p.Arg292Pro mutation acts as a pathogenic gain-of-function.
Martina Proietti Onori, Balwina Koopal, David B Everman, Jessica D Worthington, Julie R Jones, Melissa A Ploeg, Edwin Mientjes, Bregje W van Bon, Tjitske Kleefstra, Howard Schulman,[...]. Hum Mutat 2018
16
25

De novo variants in CAMK2A and CAMK2B cause neurodevelopmental disorders.
Tenpei Akita, Kazushi Aoto, Mitsuhiro Kato, Masaaki Shiina, Hiroki Mutoh, Mitsuko Nakashima, Ichiro Kuki, Shin Okazaki, Shinichi Magara, Takashi Shiihara,[...]. Ann Clin Transl Neurol 2018
38
12

De Novo Mutations in Protein Kinase Genes CAMK2A and CAMK2B Cause Intellectual Disability.
Sébastien Küry, Geeske M van Woerden, Thomas Besnard, Martina Proietti Onori, Xénia Latypova, Meghan C Towne, Megan T Cho, Trine E Prescott, Melissa A Ploeg, Stephan Sanders,[...]. Am J Hum Genet 2017
87
12




CaMKII locally encodes L-type channel activity to signal to nuclear CREB in excitation-transcription coupling.
Damian G Wheeler, Curtis F Barrett, Rachel D Groth, Parsa Safa, Richard W Tsien. J Cell Biol 2008
131
12

Long-term potentiation--a decade of progress?
R C Malenka, R A Nicoll. Science 1999
12


Analysis of the CaMKIIα and β splice-variant distribution among brain regions reveals isoform-specific differences in holoenzyme formation.
Sarah G Cook, Ashley M Bourke, Heather O'Leary, Vincent Zaegel, Erika Lasda, Janna Mize-Berge, Nidia Quillinan, Chandra L Tucker, Steven J Coultrap, Paco S Herson,[...]. Sci Rep 2018
21
14




Disruption of dendritic translation of CaMKIIalpha impairs stabilization of synaptic plasticity and memory consolidation.
Stephan Miller, Masahiro Yasuda, Jennifer K Coats, Ying Jones, Maryann E Martone, Mark Mayford. Neuron 2002
386
9

CaMKII Autophosphorylation Is Necessary for Optimal Integration of Ca2+ Signals during LTP Induction, but Not Maintenance.
Jui-Yun Chang, Paula Parra-Bueno, Tal Laviv, Erzsebet M Szatmari, Seok-Jin R Lee, Ryohei Yasuda. Neuron 2017
79
9

HDAC4 governs a transcriptional program essential for synaptic plasticity and memory.
Richard Sando, Natalia Gounko, Simon Pieraut, Lujian Liao, John Yates, Anton Maximov. Cell 2012
171
9

The CaMKII/NMDAR complex as a molecular memory.
Magdalena Sanhueza, John Lisman. Mol Brain 2013
106
9


Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.