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Times Co-cited
Similarity
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Subsets of exhausted CD8+ T cells differentially mediate tumor control and respond to checkpoint blockade.
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T Cell Factor 1-Expressing Memory-like CD8(+) T Cells Sustain the Immune Response to Chronic Viral Infections.
Daniel T Utzschneider, Mélanie Charmoy, Vijaykumar Chennupati, Laurène Pousse, Daniela Pais Ferreira, Sandra Calderon-Copete, Maxime Danilo, Francesca Alfei, Maike Hofmann, Dominik Wieland,[...]. Immunity 2016
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Defining T Cell States Associated with Response to Checkpoint Immunotherapy in Melanoma.
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Follicular CXCR5- expressing CD8(+) T cells curtail chronic viral infection.
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Clonal replacement of tumor-specific T cells following PD-1 blockade.
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TOX transcriptionally and epigenetically programs CD8+ T cell exhaustion.
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Checkpoint Blockade Immunotherapy Induces Dynamic Changes in PD-1-CD8+ Tumor-Infiltrating T Cells.
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TOX is a critical regulator of tumour-specific T cell differentiation.
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The TCF1-Bcl6 axis counteracts type I interferon to repress exhaustion and maintain T cell stemness.
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An intra-tumoral niche maintains and differentiates stem-like CD8 T cells.
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TOX reinforces the phenotype and longevity of exhausted T cells in chronic viral infection.
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Proliferating Transitory T Cells with an Effector-like Transcriptional Signature Emerge from PD-1+ Stem-like CD8+ T Cells during Chronic Infection.
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CXCR5(+) follicular cytotoxic T cells control viral infection in B cell follicles.
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TCF-1-Centered Transcriptional Network Drives an Effector versus Exhausted CD8 T Cell-Fate Decision.
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Epigenetic stability of exhausted T cells limits durability of reinvigoration by PD-1 blockade.
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Progenitor and terminal subsets of CD8+ T cells cooperate to contain chronic viral infection.
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Dysfunctional CD8 T Cells Form a Proliferative, Dynamically Regulated Compartment within Human Melanoma.
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A transcriptionally and functionally distinct PD-1+ CD8+ T cell pool with predictive potential in non-small-cell lung cancer treated with PD-1 blockade.
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Developmental Relationships of Four Exhausted CD8+ T Cell Subsets Reveals Underlying Transcriptional and Epigenetic Landscape Control Mechanisms.
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Chromatin states define tumour-specific T cell dysfunction and reprogramming.
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Tumor-Specific T Cell Dysfunction Is a Dynamic Antigen-Driven Differentiation Program Initiated Early during Tumorigenesis.
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Restoring function in exhausted CD8 T cells during chronic viral infection.
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The epigenetic landscape of T cell exhaustion.
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Defining 'T cell exhaustion'.
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Global characterization of T cells in non-small-cell lung cancer by single-cell sequencing.
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CD4+ T Cell Help Is Required for the Formation of a Cytolytic CD8+ T Cell Subset that Protects against Chronic Infection and Cancer.
Ryan Zander, David Schauder, Gang Xin, Christine Nguyen, Xiaopeng Wu, Allan Zajac, Weiguo Cui. Immunity 2019
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Single-cell RNA-seq reveals TOX as a key regulator of CD8+ T cell persistence in chronic infection.
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A human memory T cell subset with stem cell-like properties.
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Bystander CD8+ T cells are abundant and phenotypically distinct in human tumour infiltrates.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.