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Times Cited
Times Co-cited
Similarity
Inflammatory T cell responses rely on amino acid transporter ASCT2 facilitation of glutamine uptake and mTORC1 kinase activation.
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The transcription factor Myc controls metabolic reprogramming upon T lymphocyte activation.
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Glutamine blockade induces divergent metabolic programs to overcome tumor immune evasion.
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Cutting edge: distinct glycolytic and lipid oxidative metabolic programs are essential for effector and regulatory CD4+ T cell subsets.
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Metabolic Competition in the Tumor Microenvironment Is a Driver of Cancer Progression.
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HIF1alpha-dependent glycolytic pathway orchestrates a metabolic checkpoint for the differentiation of TH17 and Treg cells.
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Phosphoenolpyruvate Is a Metabolic Checkpoint of Anti-tumor T Cell Responses.
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L-Arginine Modulates T Cell Metabolism and Enhances Survival and Anti-tumor Activity.
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Glutamine uptake and metabolism are coordinately regulated by ERK/MAPK during T lymphocyte activation.
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Glutamine-dependent α-ketoglutarate production regulates the balance between T helper 1 cell and regulatory T cell generation.
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Control of amino-acid transport by antigen receptors coordinates the metabolic reprogramming essential for T cell differentiation.
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The glucose transporter Glut1 is selectively essential for CD4 T cell activation and effector function.
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PD-1 alters T-cell metabolic reprogramming by inhibiting glycolysis and promoting lipolysis and fatty acid oxidation.
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Foxp3 Reprograms T Cell Metabolism to Function in Low-Glucose, High-Lactate Environments.
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The CD28 signaling pathway regulates glucose metabolism.
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Mitochondrial Dynamics Controls T Cell Fate through Metabolic Programming.
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Serine Is an Essential Metabolite for Effector T Cell Expansion.
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Metabolic Profiling Using Stable Isotope Tracing Reveals Distinct Patterns of Glucose Utilization by Physiologically Activated CD8+ T Cells.
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LDHA-Associated Lactic Acid Production Blunts Tumor Immunosurveillance by T and NK Cells.
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Mitochondria are required for antigen-specific T cell activation through reactive oxygen species signaling.
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The energy sensor AMPK regulates T cell metabolic adaptation and effector responses in vivo.
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Etomoxir Actions on Regulatory and Memory T Cells Are Independent of Cpt1a-Mediated Fatty Acid Oxidation.
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Mitochondrial complex III is essential for suppressive function of regulatory T cells.
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Aerobic glycolysis promotes T helper 1 cell differentiation through an epigenetic mechanism.
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Inhibiting glycolytic metabolism enhances CD8+ T cell memory and antitumor function.
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Enhancing CD8+ T Cell Fatty Acid Catabolism within a Metabolically Challenging Tumor Microenvironment Increases the Efficacy of Melanoma Immunotherapy.
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Transcriptional factor ICER promotes glutaminolysis and the generation of Th17 cells.
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Mitochondrial Biogenesis and Proteome Remodeling Promote One-Carbon Metabolism for T Cell Activation.
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Memory CD8(+) T Cells Require Increased Concentrations of Acetate Induced by Stress for Optimal Function.
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Early TCR Signaling Induces Rapid Aerobic Glycolysis Enabling Distinct Acute T Cell Effector Functions.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.