Michael J Gandal, Pan Zhang, Evi Hadjimichael, Rebecca L Walker, Chao Chen, Shuang Liu, Hyejung Won, Harm van Bakel, Merina Varghese, Yongjun Wang, Annie W Shieh, Jillian Haney, Sepideh Parhami, Judson Belmont, Minsoo Kim, Patricia Moran Losada, Zenab Khan, Justyna Mleczko, Yan Xia, Rujia Dai, Daifeng Wang, Yucheng T Yang, Min Xu, Kenneth Fish, Patrick R Hof, Jonathan Warrell, Dominic Fitzgerald, Kevin White, Andrew E Jaffe, Mette A Peters, Mark Gerstein, Chunyu Liu, Lilia M Iakoucheva, Dalila Pinto, Daniel H Geschwind. Science 2018
Times Cited: 394
Times Cited: 394
Times Cited
Times Co-cited
Similarity
Gene expression elucidates functional impact of polygenic risk for schizophrenia.
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Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection.
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Comprehensive functional genomic resource and integrative model for the human brain.
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Developmental and genetic regulation of the human cortex transcriptome illuminate schizophrenia pathogenesis.
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Integrative functional genomic analysis of human brain development and neuropsychiatric risks.
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Genome-wide changes in lncRNA, splicing, and regional gene expression patterns in autism.
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Shared molecular neuropathology across major psychiatric disorders parallels polygenic overlap.
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A Single-Cell Transcriptomic Atlas of Human Neocortical Development during Mid-gestation.
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Schizophrenia risk from complex variation of complement component 4.
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Genome-wide association study identifies 30 loci associated with bipolar disorder.
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Identification of common genetic risk variants for autism spectrum disorder.
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Single-cell genomics identifies cell type-specific molecular changes in autism.
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Integration of summary data from GWAS and eQTL studies predicts complex trait gene targets.
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Integrative functional genomic analyses implicate specific molecular pathways and circuits in autism.
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Genetic identification of brain cell types underlying schizophrenia.
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Spatiotemporal gene expression trajectories reveal developmental hierarchies of the human cortex.
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Mapping DNA methylation across development, genotype and schizophrenia in the human frontal cortex.
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Transcriptome analysis reveals dysregulation of innate immune response genes and neuronal activity-dependent genes in autism.
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Coexpression networks implicate human midfetal deep cortical projection neurons in the pathogenesis of autism.
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De novo mutations in schizophrenia implicate synaptic networks.
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The road to precision psychiatry: translating genetics into disease mechanisms.
Michael J Gandal, Virpi Leppa, Hyejung Won, Neelroop N Parikshak, Daniel H Geschwind. Nat Neurosci 2016
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.