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Times Cited: 83
Times Cited: 83
Times Cited
Times Co-cited
Similarity
Alzheimer's disease: early alterations in brain DNA methylation at ANK1, BIN1, RHBDF2 and other loci.
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Tau promotes neurodegeneration through global chromatin relaxation.
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A histone acetylome-wide association study of Alzheimer's disease identifies disease-associated H3K27ac differences in the entorhinal cortex.
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Methylomic profiling implicates cortical deregulation of ANK1 in Alzheimer's disease.
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Dysregulation of the epigenetic landscape of normal aging in Alzheimer's disease.
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Single-cell transcriptomic analysis of Alzheimer's disease.
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Conserved epigenomic signals in mice and humans reveal immune basis of Alzheimer's disease.
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Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease.
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Loss of Tau protein affects the structure, transcription and repair of neuronal pericentromeric heterochromatin.
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Genome-wide meta-analysis identifies new loci and functional pathways influencing Alzheimer's disease risk.
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An integrated multi-omics approach identifies epigenetic alterations associated with Alzheimer's disease.
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DNA methylation analysis on purified neurons and glia dissects age and Alzheimer's disease-specific changes in the human cortex.
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A molecular network of the aging human brain provides insights into the pathology and cognitive decline of Alzheimer's disease.
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Integrated systems approach identifies genetic nodes and networks in late-onset Alzheimer's disease.
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Pathogenic tau-induced piRNA depletion promotes neuronal death through transposable element dysregulation in neurodegenerative tauopathies.
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Parallel profiling of DNA methylation and hydroxymethylation highlights neuropathology-associated epigenetic variation in Alzheimer's disease.
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Genetic meta-analysis of diagnosed Alzheimer's disease identifies new risk loci and implicates Aβ, tau, immunity and lipid processing.
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Epigenetic dysregulation of enhancers in neurons is associated with Alzheimer's disease pathology and cognitive symptoms.
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Ageing as a risk factor for neurodegenerative disease.
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12
Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.