Hueng-Chuen Fan, Chuan-Mu Chen, Ching-Shiang Chi, Jeng-Dau Tsai, Kuo-Liang Chiang, Yu-Kang Chang, Shinn-Zong Lin, Horng-Jyh Harn. Int J Mol Sci 2019
Times Cited: 21
Times Cited: 21
Times Cited
Times Co-cited
Similarity
TERT promoter mutations occur frequently in gliomas and a subset of tumors derived from cells with low rates of self-renewal.
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The 2016 World Health Organization Classification of Tumors of the Central Nervous System: a summary.
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Altered telomeres in tumors with ATRX and DAXX mutations.
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Alternative lengthening of telomeres renders cancer cells hypersensitive to ATR inhibitors.
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Adult infiltrating gliomas with WHO 2016 integrated diagnosis: additional prognostic roles of ATRX and TERT.
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Integrated genomic analysis identifies clinically relevant subtypes of glioblastoma characterized by abnormalities in PDGFRA, IDH1, EGFR, and NF1.
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Driver mutations in histone H3.3 and chromatin remodelling genes in paediatric glioblastoma.
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Methylation of the TERT promoter and risk stratification of childhood brain tumours: an integrative genomic and molecular study.
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Glioblastoma stem cells generate vascular pericytes to support vessel function and tumor growth.
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ATRX represses alternative lengthening of telomeres.
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ATRX immunostaining predicts IDH and H3F3A status in gliomas.
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Tumor Evolution of Glioma-Intrinsic Gene Expression Subtypes Associates with Immunological Changes in the Microenvironment.
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Distribution of EGFR amplification, combined chromosome 7 gain and chromosome 10 loss, and TERT promoter mutation in brain tumors and their potential for the reclassification of IDHwt astrocytoma to glioblastoma.
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Molecular subclasses of high-grade glioma predict prognosis, delineate a pattern of disease progression, and resemble stages in neurogenesis.
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Alternative lengthening of telomeres is enriched in, and impacts survival of TP53 mutant pediatric malignant brain tumors.
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Alternative Lengthening of Telomeres and Loss of DAXX/ATRX Expression Predicts Metastatic Disease and Poor Survival in Patients with Pancreatic Neuroendocrine Tumors.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.