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Times Cited: 180
Times Cited: 180
Times Cited
Times Co-cited
Similarity
TOX transcriptionally and epigenetically programs CD8+ T cell exhaustion.
Omar Khan, Josephine R Giles, Sierra McDonald, Sasikanth Manne, Shin Foong Ngiow, Kunal P Patel, Michael T Werner, Alexander C Huang, Katherine A Alexander, Jennifer E Wu,[...]. Nature 2019
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TOX reinforces the phenotype and longevity of exhausted T cells in chronic viral infection.
Francesca Alfei, Kristiyan Kanev, Maike Hofmann, Ming Wu, Hazem E Ghoneim, Patrick Roelli, Daniel T Utzschneider, Madlaina von Hoesslin, Jolie G Cullen, Yiping Fan,[...]. Nature 2019
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TOX is a critical regulator of tumour-specific T cell differentiation.
Andrew C Scott, Friederike Dündar, Paul Zumbo, Smita S Chandran, Christopher A Klebanoff, Mojdeh Shakiba, Prerak Trivedi, Laura Menocal, Heather Appleby, Steven Camara,[...]. Nature 2019
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TOX and TOX2 transcription factors cooperate with NR4A transcription factors to impose CD8+ T cell exhaustion.
Hyungseok Seo, Joyce Chen, Edahí González-Avalos, Daniela Samaniego-Castruita, Arundhoti Das, Yueqiang H Wang, Isaac F López-Moyado, Romain O Georges, Wade Zhang, Atsushi Onodera,[...]. Proc Natl Acad Sci U S A 2019
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Defining CD8+ T cells that provide the proliferative burst after PD-1 therapy.
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T Cell Factor 1-Expressing Memory-like CD8(+) T Cells Sustain the Immune Response to Chronic Viral Infections.
Daniel T Utzschneider, Mélanie Charmoy, Vijaykumar Chennupati, Laurène Pousse, Daniela Pais Ferreira, Sandra Calderon-Copete, Maxime Danilo, Francesca Alfei, Maike Hofmann, Dominik Wieland,[...]. Immunity 2016
Daniel T Utzschneider, Mélanie Charmoy, Vijaykumar Chennupati, Laurène Pousse, Daniela Pais Ferreira, Sandra Calderon-Copete, Maxime Danilo, Francesca Alfei, Maike Hofmann, Dominik Wieland,[...]. Immunity 2016
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Epigenetic stability of exhausted T cells limits durability of reinvigoration by PD-1 blockade.
Kristen E Pauken, Morgan A Sammons, Pamela M Odorizzi, Sasikanth Manne, Jernej Godec, Omar Khan, Adam M Drake, Zeyu Chen, Debattama R Sen, Makoto Kurachi,[...]. Science 2016
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Subsets of exhausted CD8+ T cells differentially mediate tumor control and respond to checkpoint blockade.
Brian C Miller, Debattama R Sen, Rose Al Abosy, Kevin Bi, Yamini V Virkud, Martin W LaFleur, Kathleen B Yates, Ana Lako, Kristen Felt, Girish S Naik,[...]. Nat Immunol 2019
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CD8 T Cell Exhaustion During Chronic Viral Infection and Cancer.
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TCF-1-Centered Transcriptional Network Drives an Effector versus Exhausted CD8 T Cell-Fate Decision.
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Developmental Relationships of Four Exhausted CD8+ T Cell Subsets Reveals Underlying Transcriptional and Epigenetic Landscape Control Mechanisms.
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The epigenetic landscape of T cell exhaustion.
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The TCF1-Bcl6 axis counteracts type I interferon to repress exhaustion and maintain T cell stemness.
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Intratumoral Tcf1+PD-1+CD8+ T Cells with Stem-like Properties Promote Tumor Control in Response to Vaccination and Checkpoint Blockade Immunotherapy.
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Progenitor and terminal subsets of CD8+ T cells cooperate to contain chronic viral infection.
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Molecular signature of CD8+ T cell exhaustion during chronic viral infection.
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Proliferating Transitory T Cells with an Effector-like Transcriptional Signature Emerge from PD-1+ Stem-like CD8+ T Cells during Chronic Infection.
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Molecular and cellular insights into T cell exhaustion.
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Follicular CXCR5- expressing CD8(+) T cells curtail chronic viral infection.
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NR4A transcription factors limit CAR T cell function in solid tumours.
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The transcription factor NFAT promotes exhaustion of activated CD8⁺ T cells.
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Differentiation and persistence of memory CD8(+) T cells depend on T cell factor 1.
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Defining 'T cell exhaustion'.
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Chromatin states define tumour-specific T cell dysfunction and reprogramming.
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Defining T Cell States Associated with Response to Checkpoint Immunotherapy in Melanoma.
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Restoring function in exhausted CD8 T cells during chronic viral infection.
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Network analysis reveals centrally connected genes and pathways involved in CD8+ T cell exhaustion versus memory.
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CD4+ T Cell Help Is Required for the Formation of a Cytolytic CD8+ T Cell Subset that Protects against Chronic Infection and Cancer.
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Checkpoint Blockade Immunotherapy Induces Dynamic Changes in PD-1-CD8+ Tumor-Infiltrating T Cells.
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Transcription Factor IRF4 Promotes CD8+ T Cell Exhaustion and Limits the Development of Memory-like T Cells during Chronic Infection.
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CXCR5(+) follicular cytotoxic T cells control viral infection in B cell follicles.
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Selective expansion of a subset of exhausted CD8 T cells by alphaPD-L1 blockade.
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Inflammation directs memory precursor and short-lived effector CD8(+) T cell fates via the graded expression of T-bet transcription factor.
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Early precursor T cells establish and propagate T cell exhaustion in chronic infection.
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De Novo Epigenetic Programs Inhibit PD-1 Blockade-Mediated T Cell Rejuvenation.
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An intra-tumoral niche maintains and differentiates stem-like CD8 T cells.
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Viral persistence alters CD8 T-cell immunodominance and tissue distribution and results in distinct stages of functional impairment.
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TOX is expressed by exhausted and polyfunctional human effector memory CD8+ T cells.
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Coregulation of CD8+ T cell exhaustion by multiple inhibitory receptors during chronic viral infection.
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Essential role of the Wnt pathway effector Tcf-1 for the establishment of functional CD8 T cell memory.
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Tumor-Specific T Cell Dysfunction Is a Dynamic Antigen-Driven Differentiation Program Initiated Early during Tumorigenesis.
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High-dimensional single cell analysis identifies stem-like cytotoxic CD8+ T cells infiltrating human tumors.
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TOX promotes the exhaustion of antitumor CD8+ T cells by preventing PD1 degradation in hepatocellular carcinoma.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.