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Times Cited: 132
Times Cited
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Similarity
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A 3D map of the human genome at kilobase resolution reveals principles of chromatin looping.
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Disruptions of topological chromatin domains cause pathogenic rewiring of gene-enhancer interactions.
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Targeted Degradation of CTCF Decouples Local Insulation of Chromosome Domains from Genomic Compartmentalization.
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Three-dimensional folding and functional organization principles of the Drosophila genome.
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Functional dissection of the Sox9-Kcnj2 locus identifies nonessential and instructive roles of TAD architecture.
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Two independent modes of chromatin organization revealed by cohesin removal.
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Chromatin extrusion explains key features of loop and domain formation in wild-type and engineered genomes.
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Formation of new chromatin domains determines pathogenicity of genomic duplications.
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Decreased Enhancer-Promoter Proximity Accompanying Enhancer Activation.
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Dynamic interplay between enhancer-promoter topology and gene activity.
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Multiscale 3D Genome Rewiring during Mouse Neural Development.
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Activation of proto-oncogenes by disruption of chromosome neighborhoods.
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Super-resolution chromatin tracing reveals domains and cooperative interactions in single cells.
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Chromatin Architecture Emerges during Zygotic Genome Activation Independent of Transcription.
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Live-cell imaging reveals enhancer-dependent Sox2 transcription in the absence of enhancer proximity.
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Coactivator condensation at super-enhancers links phase separation and gene control.
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Capturing chromosome conformation.
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Preformed chromatin topology assists transcriptional robustness of Shh during limb development.
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Single-nucleus Hi-C reveals unique chromatin reorganization at oocyte-to-zygote transition.
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Mediator and RNA polymerase II clusters associate in transcription-dependent condensates.
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Independence of chromatin conformation and gene regulation during Drosophila dorsoventral patterning.
Elizabeth Ing-Simmons, Roshan Vaid, Xin Yang Bing, Michael Levine, Mattias Mannervik, Juan M Vaquerizas. Nat Genet 2021
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Visualizing DNA folding and RNA in embryos at single-cell resolution.
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Functional and topological characteristics of mammalian regulatory domains.
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Developmentally regulated Shh expression is robust to TAD perturbations.
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Topologically associating domains and chromatin loops depend on cohesin and are regulated by CTCF, WAPL, and PDS5 proteins.
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Transcription Factors Activate Genes through the Phase-Separation Capacity of Their Activation Domains.
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Resolving the 3D Landscape of Transcription-Linked Mammalian Chromatin Folding.
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CRISPR Inversion of CTCF Sites Alters Genome Topology and Enhancer/Promoter Function.
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The Shh Topological Domain Facilitates the Action of Remote Enhancers by Reducing the Effects of Genomic Distances.
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Enhancer loops appear stable during development and are associated with paused polymerase.
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Chromatin architecture reorganization during stem cell differentiation.
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High-resolution TADs reveal DNA sequences underlying genome organization in flies.
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Comparative Hi-C reveals that CTCF underlies evolution of chromosomal domain architecture.
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CTCF Binding Polarity Determines Chromatin Looping.
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Active chromatin and transcription play a key role in chromosome partitioning into topologically associating domains.
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The Cohesin Release Factor WAPL Restricts Chromatin Loop Extension.
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Imaging dynamic and selective low-complexity domain interactions that control gene transcription.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.