Jiyeon Kim, Ralph J DeBerardinis. Cell Metab 2019
Times Cited: 147
Times Cited: 147
Times Cited
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Understanding the Warburg effect: the metabolic requirements of cell proliferation.
Matthew G Vander Heiden, Lewis C Cantley, Craig B Thompson. Science 2009
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Metabolic reprogramming and cancer progression.
Brandon Faubert, Ashley Solmonson, Ralph J DeBerardinis. Science 2020
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PDK1-Dependent Metabolic Reprogramming Dictates Metastatic Potential in Breast Cancer.
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Metabolic heterogeneity confers differences in melanoma metastatic potential.
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Functional genomics reveal that the serine synthesis pathway is essential in breast cancer.
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Otto Warburg's contributions to current concepts of cancer metabolism.
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The biology of cancer: metabolic reprogramming fuels cell growth and proliferation.
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GSVA: gene set variation analysis for microarray and RNA-seq data.
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Environment Impacts the Metabolic Dependencies of Ras-Driven Non-Small Cell Lung Cancer.
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Oxidative stress inhibits distant metastasis by human melanoma cells.
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7
Acetate is a bioenergetic substrate for human glioblastoma and brain metastases.
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Macropinocytosis of protein is an amino acid supply route in Ras-transformed cells.
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PGC-1α mediates mitochondrial biogenesis and oxidative phosphorylation in cancer cells to promote metastasis.
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Aerobic glycolysis: meeting the metabolic requirements of cell proliferation.
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PGC-1α Promotes Breast Cancer Metastasis and Confers Bioenergetic Flexibility against Metabolic Drugs.
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Limited Environmental Serine and Glycine Confer Brain Metastasis Sensitivity to PHGDH Inhibition.
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Proline metabolism supports metastasis formation and could be inhibited to selectively target metastasizing cancer cells.
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Glucose feeds the TCA cycle via circulating lactate.
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Phosphoglycerate dehydrogenase diverts glycolytic flux and contributes to oncogenesis.
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The metabolic profile of tumors depends on both the responsible genetic lesion and tissue type.
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An Essential Role of the Mitochondrial Electron Transport Chain in Cell Proliferation Is to Enable Aspartate Synthesis.
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From Krebs to clinic: glutamine metabolism to cancer therapy.
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Phosphoenolpyruvate Is a Metabolic Checkpoint of Anti-tumor T Cell Responses.
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Foxp3 Reprograms T Cell Metabolism to Function in Low-Glucose, High-Lactate Environments.
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Molecular Characterization and Clinical Relevance of Metabolic Expression Subtypes in Human Cancers.
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Breast Cancer-Derived Lung Metastases Show Increased Pyruvate Carboxylase-Dependent Anaplerosis.
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Adipocyte-induced CD36 expression drives ovarian cancer progression and metastasis.
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Breast cancer cells rely on environmental pyruvate to shape the metastatic niche.
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5
Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.