Juan F Burgueño, Maria T Abreu. Nat Rev Gastroenterol Hepatol 2020
Times Cited: 136
Times Cited: 136
Times Cited
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Recognition of commensal microflora by toll-like receptors is required for intestinal homeostasis.
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Gut microbial colonization orchestrates TLR2 expression, signaling and epithelial proliferation in the small intestinal mucosa.
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Toll-like receptor-4 is required for intestinal response to epithelial injury and limiting bacterial translocation in a murine model of acute colitis.
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A sentinel goblet cell guards the colonic crypt by triggering Nlrp6-dependent Muc2 secretion.
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Toll-like receptor 4 is expressed on intestinal stem cells and regulates their proliferation and apoptosis via the p53 up-regulated modulator of apoptosis.
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Colitis-associated variant of TLR2 causes impaired mucosal repair because of TFF3 deficiency.
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Toll-like receptor 2 enhances ZO-1-associated intestinal epithelial barrier integrity via protein kinase C.
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Lipopolysaccharide-Induced Increase in Intestinal Epithelial Tight Permeability Is Mediated by Toll-Like Receptor 4/Myeloid Differentiation Primary Response 88 (MyD88) Activation of Myosin Light Chain Kinase Expression.
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The gut microbiota shapes intestinal immune responses during health and disease.
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Intestinal epithelial Toll-like receptor 4 regulates goblet cell development and is required for necrotizing enterocolitis in mice.
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