Jean-Christophe Beltra, Sasikanth Manne, Mohamed S Abdel-Hakeem, Makoto Kurachi, Josephine R Giles, Zeyu Chen, Valentina Casella, Shin Foong Ngiow, Omar Khan, Yinghui Jane Huang, Patrick Yan, Kito Nzingha, Wei Xu, Ravi K Amaravadi, Xiaowei Xu, Giorgos C Karakousis, Tara C Mitchell, Lynn M Schuchter, Alexander C Huang, E John Wherry. Immunity 2020
Times Cited: 170
Times Cited: 170
Times Cited
Times Co-cited
Similarity
Subsets of exhausted CD8+ T cells differentially mediate tumor control and respond to checkpoint blockade.
Brian C Miller, Debattama R Sen, Rose Al Abosy, Kevin Bi, Yamini V Virkud, Martin W LaFleur, Kathleen B Yates, Ana Lako, Kristen Felt, Girish S Naik,[...]. Nat Immunol 2019
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TOX transcriptionally and epigenetically programs CD8+ T cell exhaustion.
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Defining CD8+ T cells that provide the proliferative burst after PD-1 therapy.
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TOX is a critical regulator of tumour-specific T cell differentiation.
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Intratumoral Tcf1+PD-1+CD8+ T Cells with Stem-like Properties Promote Tumor Control in Response to Vaccination and Checkpoint Blockade Immunotherapy.
Imran Siddiqui, Karin Schaeuble, Vijaykumar Chennupati, Silvia A Fuertes Marraco, Sandra Calderon-Copete, Daniela Pais Ferreira, Santiago J Carmona, Leonardo Scarpellino, David Gfeller, Sylvain Pradervand,[...]. Immunity 2019
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TCF-1-Centered Transcriptional Network Drives an Effector versus Exhausted CD8 T Cell-Fate Decision.
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Proliferating Transitory T Cells with an Effector-like Transcriptional Signature Emerge from PD-1+ Stem-like CD8+ T Cells during Chronic Infection.
William H Hudson, Julia Gensheimer, Masao Hashimoto, Andreas Wieland, Rajesh M Valanparambil, Peng Li, Jian-Xin Lin, Bogumila T Konieczny, Se Jin Im, Gordon J Freeman,[...]. Immunity 2019
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T Cell Factor 1-Expressing Memory-like CD8(+) T Cells Sustain the Immune Response to Chronic Viral Infections.
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CD8 T Cell Exhaustion During Chronic Viral Infection and Cancer.
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TOX reinforces the phenotype and longevity of exhausted T cells in chronic viral infection.
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Defining T Cell States Associated with Response to Checkpoint Immunotherapy in Melanoma.
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Epigenetic stability of exhausted T cells limits durability of reinvigoration by PD-1 blockade.
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Chromatin states define tumour-specific T cell dysfunction and reprogramming.
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Progenitor and terminal subsets of CD8+ T cells cooperate to contain chronic viral infection.
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Single-cell RNA-seq reveals TOX as a key regulator of CD8+ T cell persistence in chronic infection.
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CD4+ T Cell Help Is Required for the Formation of a Cytolytic CD8+ T Cell Subset that Protects against Chronic Infection and Cancer.
Ryan Zander, David Schauder, Gang Xin, Christine Nguyen, Xiaopeng Wu, Allan Zajac, Weiguo Cui. Immunity 2019
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Molecular and cellular insights into T cell exhaustion.
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An intra-tumoral niche maintains and differentiates stem-like CD8 T cells.
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The TCF1-Bcl6 axis counteracts type I interferon to repress exhaustion and maintain T cell stemness.
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Defining 'T cell exhaustion'.
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Checkpoint Blockade Immunotherapy Induces Dynamic Changes in PD-1-CD8+ Tumor-Infiltrating T Cells.
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TOX and TOX2 transcription factors cooperate with NR4A transcription factors to impose CD8+ T cell exhaustion.
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Follicular CXCR5- expressing CD8(+) T cells curtail chronic viral infection.
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The epigenetic landscape of T cell exhaustion.
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Clonal replacement of tumor-specific T cells following PD-1 blockade.
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Restoring function in exhausted CD8 T cells during chronic viral infection.
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Molecular signature of CD8+ T cell exhaustion during chronic viral infection.
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High-dimensional single cell analysis identifies stem-like cytotoxic CD8+ T cells infiltrating human tumors.
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Tumor-Specific T Cell Dysfunction Is a Dynamic Antigen-Driven Differentiation Program Initiated Early during Tumorigenesis.
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Dysfunctional CD8 T Cells Form a Proliferative, Dynamically Regulated Compartment within Human Melanoma.
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NR4A transcription factors limit CAR T cell function in solid tumours.
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De Novo Epigenetic Programs Inhibit PD-1 Blockade-Mediated T Cell Rejuvenation.
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Genetic absence of PD-1 promotes accumulation of terminally differentiated exhausted CD8+ T cells.
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Network analysis reveals centrally connected genes and pathways involved in CD8+ T cell exhaustion versus memory.
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Inflammation directs memory precursor and short-lived effector CD8(+) T cell fates via the graded expression of T-bet transcription factor.
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The PD-1/PD-L1-Checkpoint Restrains T cell Immunity in Tumor-Draining Lymph Nodes.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.