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Times Cited: 127
Times Cited
Times Co-cited
Similarity
The hallmarks of aging.
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Metformin as a Tool to Target Aging.
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T cells with dysfunctional mitochondria induce multimorbidity and premature senescence.
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Chronic inflammation (inflammaging) and its potential contribution to age-associated diseases.
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TORC1 inhibition enhances immune function and reduces infections in the elderly.
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A clinically meaningful metric of immune age derived from high-dimensional longitudinal monitoring.
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Impaired immune surveillance accelerates accumulation of senescent cells and aging.
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A sestrin-dependent Erk-Jnk-p38 MAPK activation complex inhibits immunity during aging.
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The kinase p38 activated by the metabolic regulator AMPK and scaffold TAB1 drives the senescence of human T cells.
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The Achilles' heel of senescent cells: from transcriptome to senolytic drugs.
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Benefits of Metformin in Attenuating the Hallmarks of Aging.
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A proteomic atlas of senescence-associated secretomes for aging biomarker development.
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Senolytics improve physical function and increase lifespan in old age.
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mTOR inhibition improves immune function in the elderly.
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Effects of the Insulin Sensitizer Metformin in Alzheimer Disease: Pilot Data From a Randomized Placebo-controlled Crossover Study.
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Human CD8+ EMRA T cells display a senescence-associated secretory phenotype regulated by p38 MAPK.
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Metformin inhibits mitochondrial complex I of cancer cells to reduce tumorigenesis.
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The twilight of immunity: emerging concepts in aging of the immune system.
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Cellular Senescence: Defining a Path Forward.
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Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders.
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Metformin inhibits the senescence-associated secretory phenotype by interfering with IKK/NF-κB activation.
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An essential role for senescent cells in optimal wound healing through secretion of PDGF-AA.
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A complex secretory program orchestrated by the inflammasome controls paracrine senescence.
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A senescent cell bystander effect: senescence-induced senescence.
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Senolytics decrease senescent cells in humans: Preliminary report from a clinical trial of Dasatinib plus Quercetin in individuals with diabetic kidney disease.
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Senescent cells evade immune clearance via HLA-E-mediated NK and CD8+ T cell inhibition.
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Increased risk of cognitive impairment in patients with diabetes is associated with metformin.
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Metformin, independent of AMPK, inhibits mTORC1 in a rag GTPase-dependent manner.
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Polyamines Control eIF5A Hypusination, TFEB Translation, and Autophagy to Reverse B Cell Senescence.
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p38 signaling inhibits mTORC1-independent autophagy in senescent human CD8⁺ T cells.
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Dimethylbiguanide inhibits cell respiration via an indirect effect targeted on the respiratory chain complex I.
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Naturally occurring p16(Ink4a)-positive cells shorten healthy lifespan.
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Immunosenescence and Inflamm-Aging As Two Sides of the Same Coin: Friends or Foes?
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.