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Neutrophil extracellular traps contribute to immunothrombosis in COVID-19 acute respiratory distress syndrome.
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Neutrophil extracellular traps in COVID-19.
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SARS-CoV-2-triggered neutrophil extracellular traps mediate COVID-19 pathology.
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Complement and tissue factor-enriched neutrophil extracellular traps are key drivers in COVID-19 immunothrombosis.
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Platelet activation and platelet-monocyte aggregate formation trigger tissue factor expression in patients with severe COVID-19.
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Endotheliopathy in COVID-19-associated coagulopathy: evidence from a single-centre, cross-sectional study.
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Autopsy Findings and Venous Thromboembolism in Patients With COVID-19: A Prospective Cohort Study.
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Platelets Can Associate with SARS-Cov-2 RNA and Are Hyperactivated in COVID-19.
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Clinical Characteristics of 138 Hospitalized Patients With 2019 Novel Coronavirus-Infected Pneumonia in Wuhan, China.
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Imbalanced Host Response to SARS-CoV-2 Drives Development of COVID-19.
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COVID-19: consider cytokine storm syndromes and immunosuppression.
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Dexamethasone in Hospitalized Patients with Covid-19.
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An inflammatory cytokine signature predicts COVID-19 severity and survival.
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Circulating Markers of Neutrophil Extracellular Traps Are of Prognostic Value in Patients With COVID-19.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.