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Similarity
Neutrophil extracellular traps contribute to immunothrombosis in COVID-19 acute respiratory distress syndrome.
Elizabeth A Middleton, Xue-Yan He, Frederik Denorme, Robert A Campbell, David Ng, Steven P Salvatore, Maria Mostyka, Amelia Baxter-Stoltzfus, Alain C Borczuk, Massimo Loda,[...]. Blood 2020
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Neutrophil extracellular traps in COVID-19.
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SARS-CoV-2-triggered neutrophil extracellular traps mediate COVID-19 pathology.
Flavio Protasio Veras, Marjorie Cornejo Pontelli, Camila Meirelles Silva, Juliana E Toller-Kawahisa, Mikhael de Lima, Daniele Carvalho Nascimento, Ayda Henriques Schneider, Diego Caetité, Lucas Alves Tavares, Isadora M Paiva,[...]. J Exp Med 2020
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Neutrophil extracellular traps kill bacteria.
Volker Brinkmann, Ulrike Reichard, Christian Goosmann, Beatrix Fauler, Yvonne Uhlemann, David S Weiss, Yvette Weinrauch, Arturo Zychlinsky. Science 2004
Volker Brinkmann, Ulrike Reichard, Christian Goosmann, Beatrix Fauler, Yvonne Uhlemann, David S Weiss, Yvette Weinrauch, Arturo Zychlinsky. Science 2004
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Targeting potential drivers of COVID-19: Neutrophil extracellular traps.
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Betsy J Barnes, Jose M Adrover, Amelia Baxter-Stoltzfus, Alain Borczuk, Jonathan Cools-Lartigue, James M Crawford, Juliane Daßler-Plenker, Philippe Guerci, Caroline Huynh, Jason S Knight,[...]. J Exp Med 2020
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Platelet gene expression and function in patients with COVID-19.
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Platelet activation and platelet-monocyte aggregate formation trigger tissue factor expression in patients with severe COVID-19.
Eugenio D Hottz, Isaclaudia G Azevedo-Quintanilha, Lohanna Palhinha, Lívia Teixeira, Ester A Barreto, Camila R R Pão, Cassia Righy, Sérgio Franco, Thiago M L Souza, Pedro Kurtz,[...]. Blood 2020
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Abnormal coagulation parameters are associated with poor prognosis in patients with novel coronavirus pneumonia.
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Vascular occlusion by neutrophil extracellular traps in COVID-19.
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Extracellular DNA traps promote thrombosis.
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Platelet TLR4 activates neutrophil extracellular traps to ensnare bacteria in septic blood.
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Immunothrombotic Dysregulation in COVID-19 Pneumonia Is Associated With Respiratory Failure and Coagulopathy.
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Neutrophil extracellular traps in immunity and disease.
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Systemic complement activation is associated with respiratory failure in COVID-19 hospitalized patients.
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SARS-CoV-2 binds platelet ACE2 to enhance thrombosis in COVID-19.
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Endothelial dysfunction and immunothrombosis as key pathogenic mechanisms in COVID-19.
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Neutrophil extracellular traps infiltrate the lung airway, interstitial, and vascular compartments in severe COVID-19.
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High risk of thrombosis in patients with severe SARS-CoV-2 infection: a multicenter prospective cohort study.
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The first case of COVID-19 treated with the complement C3 inhibitor AMY-101.
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Complement as a target in COVID-19?
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COVID-19: consider cytokine storm syndromes and immunosuppression.
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SARS-CoV-2 Cell Entry Depends on ACE2 and TMPRSS2 and Is Blocked by a Clinically Proven Protease Inhibitor.
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The complement system in COVID-19: friend and foe?
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Complement C3 vs C5 inhibition in severe COVID-19: Early clinical findings reveal differential biological efficacy.
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Circulating Markers of Neutrophil Extracellular Traps Are of Prognostic Value in Patients With COVID-19.
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Neutrophil elastase and myeloperoxidase regulate the formation of neutrophil extracellular traps.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.