Raffaella Buzzetti, Tiinamaija Tuomi, Didac Mauricio, Massimo Pietropaolo, Zhiguang Zhou, Paolo Pozzilli, Richard David Leslie. Diabetes 2020
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Times Cited: 1
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Tauroursodeoxycholate-Bile Acid with Chaperoning Activity: Molecular and Cellular Effects and Therapeutic Perspectives.
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Magdalena Kusaczuk. Cells 2019
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Immune and Pancreatic β Cell Interactions in Type 1 Diabetes.
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Revisiting the role of inflammation in the loss of pancreatic β-cells in T1DM.
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Decio L Eizirik, Maikel L Colli. Nat Rev Endocrinol 2020
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Teplizumab (anti-CD3 mAb) treatment preserves C-peptide responses in patients with new-onset type 1 diabetes in a randomized controlled trial: metabolic and immunologic features at baseline identify a subgroup of responders.
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Directed elimination of senescent cells by inhibition of BCL-W and BCL-XL.
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Differentiation of Diabetes by Pathophysiology, Natural History, and Prognosis.
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Mitochondrial Reactive Oxygen Species and Type 1 Diabetes.
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A combined risk score enhances prediction of type 1 diabetes among susceptible children.
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Protection from autoimmune diabetes and T-cell lymphoproliferation induced by FasL mutation are differentially regulated and can be uncoupled pharmacologically.
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Targeting senescent cells in translational medicine.
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PDL1 is expressed in the islets of people with type 1 diabetes and is up-regulated by interferons-α and-γ via IRF1 induction.
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Proinsulin Secretion Is a Persistent Feature of Type 1 Diabetes.
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Persistence of Pancreatic Insulin mRNA Expression and Proinsulin Protein in Type 1 Diabetes Pancreata.
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A review on insulin trafficking and exocytosis.
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Reducing Senescent Cell Burden in Aging and Disease.
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Phase 1 Study of Molibresib (GSK525762), a Bromodomain and Extra-Terminal Domain Protein Inhibitor, in NUT Carcinoma and Other Solid Tumors.
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Randomized 52-week Phase 2 Trial of Albiglutide Versus Placebo in Adult Patients With Newly Diagnosed Type 1 Diabetes.
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The importance of the Non Obese Diabetic (NOD) mouse model in autoimmune diabetes.
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The remission phase in type 1 diabetes: Changing epidemiology, definitions, and emerging immuno-metabolic mechanisms.
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Islet β-cell endoplasmic reticulum stress precedes the onset of type 1 diabetes in the nonobese diabetic mouse model.
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Type 1 diabetes mellitus as a disease of the β-cell (do not blame the immune system?).
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Cellular senescence drives age-dependent hepatic steatosis.
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Virgin Beta Cells Persist throughout Life at a Neogenic Niche within Pancreatic Islets.
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