Roxane Crabé, Franck Aimond, Philippe Gosset, Frédérique Scamps, Cédric Raoul. Cells 2020
Times Cited: 7
Times Cited: 7
Times Cited
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Ubiquitinated TDP-43 in frontotemporal lobar degeneration and amyotrophic lateral sclerosis.
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Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis.
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Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS.
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TDP-43 mutant transgenic mice develop features of ALS and frontotemporal lobar degeneration.
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Motor neurons in Cu/Zn superoxide dismutase-deficient mice develop normally but exhibit enhanced cell death after axonal injury.
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TDP-43 mediates degeneration in a novel Drosophila model of disease caused by mutations in VCP/p97.
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Poly(ADP-Ribose) Prevents Pathological Phase Separation of TDP-43 by Promoting Liquid Demixing and Stress Granule Localization.
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Human SOD1 ALS Mutations in a Drosophila Knock-In Model Cause Severe Phenotypes and Reveal Dosage-Sensitive Gain- and Loss-of-Function Components.
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Motor neuron apoptosis and neuromuscular junction perturbation are prominent features in a Drosophila model of Fus-mediated ALS.
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Differential Toxicity of Nuclear RNA Foci versus Dipeptide Repeat Proteins in a Drosophila Model of C9ORF72 FTD/ALS.
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C9orf72 is required for proper macrophage and microglial function in mice.
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Prion-like Mechanism in Amyotrophic Lateral Sclerosis: are Protein Aggregates the Key?
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Antisense proline-arginine RAN dipeptides linked to C9ORF72-ALS/FTD form toxic nuclear aggregates that initiate in vitro and in vivo neuronal death.
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The genome sequence of Drosophila melanogaster.
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Loss of function of C9orf72 causes motor deficits in a zebrafish model of amyotrophic lateral sclerosis.
Sorana Ciura, Serena Lattante, Isabelle Le Ber, Morwena Latouche, Hervé Tostivint, Alexis Brice, Edor Kabashi. Ann Neurol 2013
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The ALS gene FUS regulates synaptic transmission at the Drosophila neuromuscular junction.
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Severe respiratory changes at end stage in a FUS-induced disease state in adult rats.
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Knockdown of transactive response DNA-binding protein (TDP-43) downregulates histone deacetylase 6.
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The unfolded protein response: integrating stress signals through the stress sensor IRE1α.
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Motor neuron expression of the voltage-gated calcium channel cacophony restores locomotion defects in a Drosophila, TDP-43 loss of function model of ALS.
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Expanded GGGGCC repeat RNA associated with amyotrophic lateral sclerosis and frontotemporal dementia causes neurodegeneration.
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The C9orf72 GGGGCC repeat is translated into aggregating dipeptide-repeat proteins in FTLD/ALS.
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Suppression of C9orf72 RNA repeat-induced neurotoxicity by the ALS-associated RNA-binding protein Zfp106.
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Highly efficient cell-type-specific gene inactivation reveals a key function for the Drosophila FUS homolog cabeza in neurons.
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ALS Genetics, Mechanisms, and Therapeutics: Where Are We Now?
Rita Mejzini, Loren L Flynn, Ianthe L Pitout, Sue Fletcher, Steve D Wilton, P Anthony Akkari. Front Neurosci 2019
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Depletion of TDP-43 affects Drosophila motoneurons terminal synapsis and locomotive behavior.
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Accumulation of insoluble forms of FUS protein correlates with toxicity in Drosophila.
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Exome sequencing reveals VCP mutations as a cause of familial ALS.
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Genetic link between Cabeza, a Drosophila homologue of Fused in Sarcoma (FUS), and the EGFR signaling pathway.
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Deletion of C9ORF72 results in motor neuron degeneration and stress sensitivity in C. elegans.
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A drosophila model for amyotrophic lateral sclerosis reveals motor neuron damage by human SOD1.
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Knockdown of the Drosophila fused in sarcoma (FUS) homologue causes deficient locomotive behavior and shortening of motoneuron terminal branches.
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Expression of human FUS protein in Drosophila leads to progressive neurodegeneration.
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Genetic strategies to tackle neurological diseases in fruit flies.
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A comparative bioinformatic analysis of C9orf72.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.