Megha Amar, Akula Bala Pramod, Nam-Kyung Yu, Victor Munive Herrera, Lily R Qiu, Patricia Moran-Losada, Pan Zhang, Cleber A Trujillo, Jacob Ellegood, Jorge Urresti, Kevin Chau, Jolene Diedrich, Jiaye Chen, Jessica Gutierrez, Jonathan Sebat, Dhakshin Ramanathan, Jason P Lerch, John R Yates, Alysson R Muotri, Lilia M Iakoucheva. Mol Psychiatry 2021
Times Cited: 3
Times Cited: 3
Times Cited
Times Co-cited
Similarity
Spatiotemporal 16p11.2 protein network implicates cortical late mid-fetal brain development and KCTD13-Cul3-RhoA pathway in psychiatric diseases.
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Inhibition of SRGAP2 function by its human-specific paralogs induces neoteny during spine maturation.
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Proneural transcription factors regulate different steps of cortical neuron migration through Rnd-mediated inhibition of RhoA signaling.
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Spatial and temporal mapping of de novo mutations in schizophrenia to a fetal prefrontal cortical network.
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Complex Oscillatory Waves Emerging from Cortical Organoids Model Early Human Brain Network Development.
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Pervasive genetic interactions modulate neurodevelopmental defects of the autism-associated 16p11.2 deletion in Drosophila melanogaster.
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A novel RhoA/ROCK-CPI-17-MEF2C signaling pathway regulates vascular smooth muscle cell gene expression.
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A radial glia-specific role of RhoA in double cortex formation.
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Human iPSC-Derived Cerebral Organoids Model Cellular Features of Lissencephaly and Reveal Prolonged Mitosis of Outer Radial Glia.
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Kctd13 deletion reduces synaptic transmission via increased RhoA.
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Prickle1 promotes focal adhesion disassembly in cooperation with the CLASP-LL5β complex in migrating cells.
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Genes influenced by MEF2C contribute to neurodevelopmental disease via gene expression changes that affect multiple types of cortical excitatory neurons.
Donna Cosgrove, Laura Whitton, Laura Fahey, Pilib Ó Broin, Gary Donohoe, Derek W Morris. Hum Mol Genet 2021
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CNVs: harbingers of a rare variant revolution in psychiatric genetics.
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Regulation of cerebral cortex development by Rho GTPases: insights from in vivo studies.
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Building Models of Brain Disorders with Three-Dimensional Organoids.
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CRISPR/Cas9-mediated heterozygous knockout of the autism gene CHD8 and characterization of its transcriptional networks in cerebral organoids derived from iPS cells.
Ping Wang, Ryan Mokhtari, Erika Pedrosa, Michael Kirschenbaum, Can Bayrak, Deyou Zheng, Herbert M Lachman. Mol Autism 2017
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Wnt signaling in development and disease.
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Zika virus impairs growth in human neurospheres and brain organoids.
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Association between microdeletion and microduplication at 16p11.2 and autism.
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Functional impact of global rare copy number variation in autism spectrum disorders.
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Insights into Autism Spectrum Disorder Genomic Architecture and Biology from 71 Risk Loci.
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Behavioral abnormalities and circuit defects in the basal ganglia of a mouse model of 16p11.2 deletion syndrome.
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The neurodegenerative disease protein ataxin-1 antagonizes the neuronal survival function of myocyte enhancer factor-2.
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Wnt signaling regulates neuronal differentiation of cortical intermediate progenitors.
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G protein-coupled receptor 56 and collagen III, a receptor-ligand pair, regulates cortical development and lamination.
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FOXG1-Dependent Dysregulation of GABA/Glutamate Neuron Differentiation in Autism Spectrum Disorders.
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The 16p11.2 deletion mouse model of autism exhibits altered cortical progenitor proliferation and brain cytoarchitecture linked to the ERK MAPK pathway.
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Cerebral organoids model human brain development and microcephaly.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.