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Times Cited: 17
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Times Co-cited
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The whole-genome landscape of medulloblastoma subtypes.
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Intertumoral Heterogeneity within Medulloblastoma Subgroups.
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Molecular subgroups of medulloblastoma: the current consensus.
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Michael D Taylor, Paul A Northcott, Andrey Korshunov, Marc Remke, Yoon-Jae Cho, Steven C Clifford, Charles G Eberhart, D Williams Parsons, Stefan Rutkowski, Amar Gajjar,[...]. Acta Neuropathol 2012
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Subgroup-specific structural variation across 1,000 medulloblastoma genomes.
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Subgroup-specific prognostic implications of TP53 mutation in medulloblastoma.
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Subtypes of medulloblastoma have distinct developmental origins.
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Genome sequencing of SHH medulloblastoma predicts genotype-related response to smoothened inhibition.
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Marcel Kool, David T W Jones, Natalie Jäger, Paul A Northcott, Trevor J Pugh, Volker Hovestadt, Rosario M Piro, L Adriana Esparza, Shirley L Markant, Marc Remke,[...]. Cancer Cell 2014
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Resolving medulloblastoma cellular architecture by single-cell genomics.
Volker Hovestadt, Kyle S Smith, Laure Bihannic, Mariella G Filbin, McKenzie L Shaw, Alicia Baumgartner, John C DeWitt, Andrew Groves, Lisa Mayr, Hannah R Weisman,[...]. Nature 2019
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Novel molecular subgroups for clinical classification and outcome prediction in childhood medulloblastoma: a cohort study.
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Pediatric and adult sonic hedgehog medulloblastomas are clinically and molecularly distinct.
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The 2016 World Health Organization Classification of Tumors of the Central Nervous System: a summary.
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Novel mutations target distinct subgroups of medulloblastoma.
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Medulloblastoma comprises four distinct molecular variants.
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scRNA-seq in medulloblastoma shows cellular heterogeneity and lineage expansion support resistance to SHH inhibitor therapy.
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Medulloblastoma can be initiated by deletion of Patched in lineage-restricted progenitors or stem cells.
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Medulloblastoma in the age of molecular subgroups: a review.
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Recurrence patterns across medulloblastoma subgroups: an integrated clinical and molecular analysis.
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Aberrant ERBB4-SRC Signaling as a Hallmark of Group 4 Medulloblastoma Revealed by Integrative Phosphoproteomic Profiling.
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Enhancer hijacking activates GFI1 family oncogenes in medulloblastoma.
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Second-generation molecular subgrouping of medulloblastoma: an international meta-analysis of Group 3 and Group 4 subtypes.
Tanvi Sharma, Edward C Schwalbe, Daniel Williamson, Martin Sill, Volker Hovestadt, Martin Mynarek, Stefan Rutkowski, Giles W Robinson, Amar Gajjar, Florence Cavalli,[...]. Acta Neuropathol 2019
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Risk stratification of childhood medulloblastoma in the molecular era: the current consensus.
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Medulloblastomics: the end of the beginning.
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Integrative genomic analysis of medulloblastoma identifies a molecular subgroup that drives poor clinical outcome.
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Integrated genomics identifies five medulloblastoma subtypes with distinct genetic profiles, pathway signatures and clinicopathological features.
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Clinical and mutational profiles of adult medulloblastoma groups.
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Recurrent noncoding U1 snRNA mutations drive cryptic splicing in SHH medulloblastoma.
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Adult medulloblastoma comprises three major molecular variants.
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DNA methylation-based classification of central nervous system tumours.
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Medulloblastoma: clinicopathological correlates of SHH, WNT, and non-SHH/WNT molecular subgroups.
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Childhood cerebellar tumours mirror conserved fetal transcriptional programs.
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Prognostic value of medulloblastoma extent of resection after accounting for molecular subgroup: a retrospective integrated clinical and molecular analysis.
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The 2021 WHO Classification of Tumors of the Central Nervous System: a summary.
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Quiescent sox2(+) cells drive hierarchical growth and relapse in sonic hedgehog subgroup medulloblastoma.
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Prognostic relevance of tumor-infiltrating lymphocytes and immune checkpoints in pediatric medulloblastoma.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.