Radhia Kacher, François-Xavier Lejeune, Sandrine Noël, Cécile Cazeneuve, Alexis Brice, Sandrine Humbert, Alexandra Durr. Elife 2021
Times Cited: 10
Times Cited: 10
Times Cited
Times Co-cited
Similarity
60
Somatic expansion of the Huntington's disease CAG repeat in the brain is associated with an earlier age of disease onset.
Meera Swami, Audrey E Hendricks, Tammy Gillis, Tiffany Massood, Jayalakshmi Mysore, Richard H Myers, Vanessa C Wheeler. Hum Mol Genet 2009
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Dramatic tissue-specific mutation length increases are an early molecular event in Huntington disease pathogenesis.
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DNA repair pathways underlie a common genetic mechanism modulating onset in polyglutamine diseases.
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A genetic association study of glutamine-encoding DNA sequence structures, somatic CAG expansion, and DNA repair gene variants, with Huntington disease clinical outcomes.
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Genetic and Functional Analyses Point to FAN1 as the Source of Multiple Huntington Disease Modifier Effects.
Kyung-Hee Kim, Eun Pyo Hong, Jun Wan Shin, Michael J Chao, Jacob Loupe, Tammy Gillis, Jayalakshmi S Mysore, Peter Holmans, Lesley Jones, Michael Orth,[...]. Am J Hum Genet 2020
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MSH3 modifies somatic instability and disease severity in Huntington's and myotonic dystrophy type 1.
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Patterns of CAG repeat instability in the central nervous system and periphery in Huntington's disease and in spinocerebellar ataxia type 1.
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Ricardo Mouro Pinto, Larissa Arning, James V Giordano, Pedram Razghandi, Marissa A Andrew, Tammy Gillis, Kevin Correia, Jayalakshmi S Mysore, Debora-M Grote Urtubey, Constanze R Parwez,[...]. Hum Mol Genet 2020
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Length-dependent gametic CAG repeat instability in the Huntington's disease knock-in mouse.
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Choosing an animal model for the study of Huntington's disease.
Mahmoud A Pouladi, A Jennifer Morton, Michael R Hayden. Nat Rev Neurosci 2013
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N-terminal Huntingtin Knock-In Mice: Implications of Removing the N-terminal Region of Huntingtin for Therapy.
Xudong Liu, Chuan-En Wang, Yan Hong, Ting Zhao, Guohao Wang, Marta A Gaertig, Miao Sun, Shihua Li, Xiao-Jiang Li. PLoS Genet 2016
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Neurological abnormalities in a knock-in mouse model of Huntington's disease.
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Selective striatal neuronal loss in a YAC128 mouse model of Huntington disease.
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Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice.
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Full-length human mutant huntingtin with a stable polyglutamine repeat can elicit progressive and selective neuropathogenesis in BACHD mice.
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RAN Translation in Huntington Disease.
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Ablation of huntingtin in adult neurons is nondeleterious but its depletion in young mice causes acute pancreatitis.
Guohao Wang, Xudong Liu, Marta A Gaertig, Shihua Li, Xiao-Jiang Li. Proc Natl Acad Sci U S A 2016
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Mutant Huntingtin Disrupts the Nuclear Pore Complex.
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A Huntingtin Knockin Pig Model Recapitulates Features of Selective Neurodegeneration in Huntington's Disease.
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Time course of early motor and neuropathological anomalies in a knock-in mouse model of Huntington's disease with 140 CAG repeats.
Liliana B Menalled, Jessica D Sison, Ioannis Dragatsis, Scott Zeitlin, Marie-Françoise Chesselet. J Comp Neurol 2003
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Generation of an isogenic, gene-corrected control cell line of the spinocerebellar ataxia type 2 patient-derived iPSC line H266.
Adele G Marthaler, Alisa Tubsuwan, Benjamin Schmid, Ulla B Poulsen, Alexander F Engelbrecht, Ulrike A Mau-Holzmann, Poul Hyttel, Troels T Nielsen, Jørgen E Nielsen, Bjørn Holst. Stem Cell Res 2016
Adele G Marthaler, Alisa Tubsuwan, Benjamin Schmid, Ulla B Poulsen, Alexander F Engelbrecht, Ulrike A Mau-Holzmann, Poul Hyttel, Troels T Nielsen, Jørgen E Nielsen, Bjørn Holst. Stem Cell Res 2016
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CRISPR/Cas9-Targeted Deletion of Polyglutamine in Spinocerebellar Ataxia Type 3-Derived Induced Pluripotent Stem Cells.
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Generation of an isogenic, gene-corrected control cell line of the spinocerebellar ataxia type 2 patient-derived iPSC line H196.
Adele G Marthaler, Benjamin Schmid, Alisa Tubsuwan, Ulla B Poulsen, Alexander F Engelbrecht, Ulrike A Mau-Holzmann, Poul Hyttel, Jørgen E Nielsen, Troels T Nielsen, Bjørn Holst. Stem Cell Res 2016
Adele G Marthaler, Benjamin Schmid, Alisa Tubsuwan, Ulla B Poulsen, Alexander F Engelbrecht, Ulrike A Mau-Holzmann, Poul Hyttel, Jørgen E Nielsen, Troels T Nielsen, Bjørn Holst. Stem Cell Res 2016
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Targeting Huntingtin Expression in Patients with Huntington's Disease.
Sarah J Tabrizi, Blair R Leavitt, G Bernhard Landwehrmeyer, Edward J Wild, Carsten Saft, Roger A Barker, Nick F Blair, David Craufurd, Josef Priller, Hugh Rickards,[...]. N Engl J Med 2019
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Genetics, Mechanisms, and Therapeutic Progress in Polyglutamine Spinocerebellar Ataxias.
Ronald A M Buijsen, Lodewijk J A Toonen, Sarah L Gardiner, Willeke M C van Roon-Mom. Neurotherapeutics 2019
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Generation of an isogenic, gene-corrected control cell line of the spinocerebellar ataxia type 2 patient-derived iPSC line H271.
Adele G Marthaler, Benjamin Schmid, Alisa Tubsuwan, Ulla B Poulsen, Alexander F Engelbrecht, Ulrike A Mau-Holzmann, Poul Hyttel, Jørgen E Nielsen, Troels T Nielsen, Bjørn Holst. Stem Cell Res 2016
Adele G Marthaler, Benjamin Schmid, Alisa Tubsuwan, Ulla B Poulsen, Alexander F Engelbrecht, Ulrike A Mau-Holzmann, Poul Hyttel, Jørgen E Nielsen, Troels T Nielsen, Bjørn Holst. Stem Cell Res 2016
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The central role of DNA damage and repair in CAG repeat diseases.
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Chromosomal instability during neurogenesis in Huntington's disease.
Albert Ruzo, Gist F Croft, Jakob J Metzger, Szilvia Galgoczi, Lauren J Gerber, Cecilia Pellegrini, Hanbin Wang, Maria Fenner, Stephanie Tse, Adam Marks,[...]. Development 2018
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Generation of New Isogenic Models of Huntington's Disease Using CRISPR-Cas9 Technology.
Magdalena Dabrowska, Agata Ciolak, Emilia Kozlowska, Agnieszka Fiszer, Marta Olejniczak. Int J Mol Sci 2020
Magdalena Dabrowska, Agata Ciolak, Emilia Kozlowska, Agnieszka Fiszer, Marta Olejniczak. Int J Mol Sci 2020
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Characterization of Human Huntington's Disease Cell Model from Induced Pluripotent Stem Cells.
Ningzhe Zhang, Mahru C An, Daniel Montoro, Lisa M Ellerby. PLoS Curr 2010
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Genetic correction of Huntington's disease phenotypes in induced pluripotent stem cells.
Mahru C An, Ningzhe Zhang, Gary Scott, Daniel Montoro, Tobias Wittkop, Sean Mooney, Simon Melov, Lisa M Ellerby. Cell Stem Cell 2012
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Faulty neuronal determination and cell polarization are reverted by modulating HD early phenotypes.
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Breaking the code of DNA binding specificity of TAL-type III effectors.
Jens Boch, Heidi Scholze, Sebastian Schornack, Angelika Landgraf, Simone Hahn, Sabine Kay, Thomas Lahaye, Anja Nickstadt, Ulla Bonas. Science 2009
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Lack of RAN-mediated toxicity in Huntington's disease knock-in mice.
Su Yang, Huiming Yang, Luoxiu Huang, Luxiao Chen, Zhaohui Qin, Shihua Li, Xiao-Jiang Li. Proc Natl Acad Sci U S A 2020
Su Yang, Huiming Yang, Luoxiu Huang, Luxiao Chen, Zhaohui Qin, Shihua Li, Xiao-Jiang Li. Proc Natl Acad Sci U S A 2020
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Reversal of Phenotypic Abnormalities by CRISPR/Cas9-Mediated Gene Correction in Huntington Disease Patient-Derived Induced Pluripotent Stem Cells.
Xiaohong Xu, Yilin Tay, Bernice Sim, Su-In Yoon, Yihui Huang, Jolene Ooi, Kagistia Hana Utami, Amin Ziaei, Bryan Ng, Carola Radulescu,[...]. Stem Cell Reports 2017
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A programmable dual-RNA-guided DNA endonuclease in adaptive bacterial immunity.
Martin Jinek, Krzysztof Chylinski, Ines Fonfara, Michael Hauer, Jennifer A Doudna, Emmanuelle Charpentier. Science 2012
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Disease-specific induced pluripotent stem cells.
In-Hyun Park, Natasha Arora, Hongguang Huo, Nimet Maherali, Tim Ahfeldt, Akiko Shimamura, M William Lensch, Chad Cowan, Konrad Hochedlinger, George Q Daley. Cell 2008
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Molecular mechanisms underlying nucleotide repeat expansion disorders.
Indranil Malik, Chase P Kelley, Eric T Wang, Peter K Todd. Nat Rev Mol Cell Biol 2021
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A Human Induced Pluripotent Stem Cell-Derived Isogenic Model of Huntington's Disease Based on Neuronal Cells Has Several Relevant Phenotypic Abnormalities.
Tuyana Malankhanova, Lyubov Suldina, Elena Grigor'eva, Sergey Medvedev, Julia Minina, Ksenia Morozova, Elena Kiseleva, Suren Zakian, Anastasia Malakhova. J Pers Med 2020
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The first reported generation of several induced pluripotent stem cell lines from homozygous and heterozygous Huntington's disease patients demonstrates mutation related enhanced lysosomal activity.
Stefano Camnasio, Alessia Delli Carri, Angelo Lombardo, Iwona Grad, Caterina Mariotti, Alessia Castucci, Björn Rozell, Pietro Lo Riso, Valentina Castiglioni, Chiara Zuccato,[...]. Neurobiol Dis 2012
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20
Unbiased Profiling of Isogenic Huntington Disease hPSC-Derived CNS and Peripheral Cells Reveals Strong Cell-Type Specificity of CAG Length Effects.
Jolene Ooi, Sarah R Langley, Xiaohong Xu, Kagistia H Utami, Bernice Sim, Yihui Huang, Nathan P Harmston, Yi Lin Tay, Amin Ziaei, Ruizhu Zeng,[...]. Cell Rep 2019
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Modifiers of Somatic Repeat Instability in Mouse Models of Friedreich Ataxia and the Fragile X-Related Disorders: Implications for the Mechanism of Somatic Expansion in Huntington's Disease.
Xiaonan Zhao, Daman Kumari, Carson J Miller, Geum-Yi Kim, Bruce Hayward, Antonia G Vitalo, Ricardo Mouro Pinto, Karen Usdin. J Huntingtons Dis 2021
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Gametic and somatic tissue-specific heterogeneity of the expanded SCA1 CAG repeat in spinocerebellar ataxia type 1.
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MutLγ promotes repeat expansion in a Fragile X mouse model while EXO1 is protective.
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Somatic and gonadal mosaicism of the Huntington disease gene CAG repeat in brain and sperm.
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Longitudinal increases in somatic mosaicism of the expanded CTG repeat in myotonic dystrophy type 1 are associated with variation in age-at-onset.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.