Mijin Kim, Bo Hyun Kim. Endocrinol Metab (Seoul) 2021
Times Cited: 4
Times Cited: 4
Times Cited
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Similarity
Structural basis of resistance of mutant RET protein-tyrosine kinase to its inhibitors nintedanib and vandetanib.
Simon S Terzyan, Tao Shen, Xuan Liu, Qingling Huang, Peng Teng, Mi Zhou, Frank Hilberg, Jianfeng Cai, Blaine H M Mooers, Jie Wu. J Biol Chem 2019
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Ginsenoside Rg3 induces ginsenoside Rb1-comparable cardioprotective effects independent of reducing blood pressure in spontaneously hypertensive rats.
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A molecular basis for cardiac arrhythmia: HERG mutations cause long QT syndrome.
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Computational determination of hERG-related cardiotoxicity of drug candidates.
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The lack of target specificity of small molecule anticancer kinase inhibitors is correlated with their ability to damage myocytes in vitro.
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Xiaolai Yang, Jian Zou, Hongyi Cai, Xiaoling Huang, Xiongfei Yang, Dexi Guo, Yongxiao Cao. Biomed Pharmacother 2017
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hERG toxicity assessment: Useful guidelines for drug design.
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Increase in CO2 levels by upregulating late sodium current is proarrhythmic in the heart.
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hERG 1b is critical for human cardiac repolarization.
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Selective use of vandetanib in the treatment of thyroid cancer.
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Anticancer effects of ginsenoside Rg3 (Review).
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c-RET molecule in malignant melanoma from oncogenic RET-carrying transgenic mice and human cell lines.
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A secondary RET mutation in the activation loop conferring resistance to vandetanib.
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[Detection of novel driver mutations in liquid biopsy: case report of a RET-positive lung adenocarcinoma treated with pralsetinib.]
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RET inhibition in novel patient-derived models of RET-fusion positive lung adenocarcinoma reveals a role for MYC upregulation.
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