Jesmin Akter, Yutaka Katai, Parvin Sultana, Hisanori Takenobu, Masayuki Haruta, Ryuichi P Sugino, Kyosuke Mukae, Shunpei Satoh, Tomoko Wada, Miki Ohira, Kiyohiro Ando, Takehiko Kamijo. Oncogenesis 2021
Times Cited: 1
Times Cited: 1
Times Cited
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ATRX represses alternative lengthening of telomeres.
Christine E Napier, Lily I Huschtscha, Adam Harvey, Kylie Bower, Jane R Noble, Eric A Hendrickson, Roger R Reddel. Oncotarget 2015
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TERRA transcription destabilizes telomere integrity to initiate break-induced replication in human ALT cells.
Bruno Silva, Rajika Arora, Silvia Bione, Claus M Azzalin. Nat Commun 2021
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FANCD2 limits BLM-dependent telomere instability in the alternative lengthening of telomeres pathway.
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RAD52 and SLX4 act nonepistatically to ensure telomere stability during alternative telomere lengthening.
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Break-induced telomere synthesis underlies alternative telomere maintenance.
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Alternative lengthening of telomeres is a self-perpetuating process in ALT-associated PML bodies.
Jia-Min Zhang, Marie-Michelle Genois, Jian Ouyang, Li Lan, Lee Zou. Mol Cell 2021
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G-quadruplex Stabilization Fuels the ALT Pathway in ALT-positive Osteosarcoma Cells.
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FANCM, BRCA1, and BLM cooperatively resolve the replication stress at the ALT telomeres.
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Brca2 abrogation engages with the alternative lengthening of telomeres via break-induced replication.
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Loss of ATRX Suppresses Resolution of Telomere Cohesion to Control Recombination in ALT Cancer Cells.
Mahesh Ramamoorthy, Susan Smith. Cancer Cell 2015
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The Role of ATRX in the Alternative Lengthening of Telomeres (ALT) Phenotype.
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SLX4IP Antagonizes Promiscuous BLM Activity during ALT Maintenance.
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Telomere Length Determines TERRA and R-Loop Regulation through the Cell Cycle.
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Nuclear receptors regulate alternative lengthening of telomeres through a novel noncanonical FANCD2 pathway.
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Differential requirements for DNA repair proteins in immortalized cell lines using alternative lengthening of telomere mechanisms.
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RNaseH1 regulates TERRA-telomeric DNA hybrids and telomere maintenance in ALT tumour cells.
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Noncovalent interactions with SUMO and ubiquitin orchestrate distinct functions of the SLX4 complex in genome maintenance.
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ATRX loss induces telomere dysfunction and necessitates induction of alternative lengthening of telomeres during human cell immortalization.
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The macroH2A1.2 histone variant links ATRX loss to alternative telomere lengthening.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.