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Times Cited: 654
Times Cited: 654
Times Cited
Times Co-cited
Similarity
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Massive genomic rearrangement acquired in a single catastrophic event during cancer development.
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BET bromodomain inhibition as a therapeutic strategy to target c-Myc.
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Drugging MYCN through an allosteric transition in Aurora kinase A.
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Extrachromosomal driver mutations in glioblastoma and low-grade glioma.
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Molecular structure of double-minute chromosomes bearing amplified copies of the epidermal growth factor receptor gene in gliomas.
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Extrachromosomal microDNAs and chromosomal microdeletions in normal tissues.
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Discoveries of Extrachromosomal Circles of DNA in Normal and Tumor Cells.
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Inhibiting homologous recombination decreases extrachromosomal amplification but has no effect on intrachromosomal amplification in methotrexate-resistant colon cancer cells.
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Enhancer hijacking determines extrachromosomal circular MYCN amplicon architecture in neuroblastoma.
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10
10
Max: a helix-loop-helix zipper protein that forms a sequence-specific DNA-binding complex with Myc.
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Targeting MYCN-Driven Transcription By BET-Bromodomain Inhibition.
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Extrachromosomal circular DNA is common in yeast.
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Henrik D Møller, Lance Parsons, Tue S Jørgensen, David Botstein, Birgitte Regenberg. Proc Natl Acad Sci U S A 2015
10
Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.