A citation-based method for searching scientific literature

C Deng, A Wynshaw-Boris, F Zhou, A Kuo, P Leder. Cell 1996
Times Cited: 834







List of co-cited articles
981 articles co-cited >1



Times Cited
  Times     Co-cited
Similarity


Skeletal overgrowth and deafness in mice lacking fibroblast growth factor receptor 3.
J S Colvin, B A Bohne, G W Harding, D G McEwen, D M Ornitz. Nat Genet 1996
687
41

Mutations in the transmembrane domain of FGFR3 cause the most common genetic form of dwarfism, achondroplasia.
R Shiang, L M Thompson, Y Z Zhu, D M Church, T J Fielder, M Bocian, S T Winokur, J J Wasmuth. Cell 1994
916
27


Gly369Cys mutation in mouse FGFR3 causes achondroplasia by affecting both chondrogenesis and osteogenesis.
L Chen, R Adar, X Yang, E O Monsonego, C Li, P V Hauschka, A Yayon, C X Deng. J Clin Invest 1999
188
20

Mutations in the gene encoding fibroblast growth factor receptor-3 in achondroplasia.
F Rousseau, J Bonaventure, L Legeai-Mallet, A Pelet, J M Rozet, P Maroteaux, M Le Merrer, A Munnich. Nature 1994
649
17

Achondroplasia.
William A Horton, Judith G Hall, Jacqueline T Hecht. Lancet 2007
268
17

The Fibroblast Growth Factor signaling pathway.
David M Ornitz, Nobuyuki Itoh. Wiley Interdiscip Rev Dev Biol 2015
845
17


A novel mutation in FGFR3 causes camptodactyly, tall stature, and hearing loss (CATSHL) syndrome.
Reha M Toydemir, Anna E Brassington, Pinar Bayrak-Toydemir, Patrycja A Krakowiak, Lynn B Jorde, Frank G Whitby, Nicola Longo, David H Viskochil, John C Carey, Michael J Bamshad. Am J Hum Genet 2006
85
18


Development of the endochondral skeleton.
Fanxin Long, David M Ornitz. Cold Spring Harb Perspect Biol 2013
287
15


Gain-of-function mutation in FGFR3 in mice leads to decreased bone mass by affecting both osteoblastogenesis and osteoclastogenesis.
Nan Su, Qidi Sun, Can Li, Xiumin Lu, Huabing Qi, Siyu Chen, Jing Yang, Xiaolan Du, Ling Zhao, Qifen He,[...]. Hum Mol Genet 2010
64
23



A neonatal lethal mutation in FGFR3 uncouples proliferation and differentiation of growth plate chondrocytes in embryos.
T Iwata, L Chen, C Li, D A Ovchinnikov, R R Behringer, C A Francomano, C X Deng. Hum Mol Genet 2000
157
14

A mouse model for achondroplasia produced by targeting fibroblast growth factor receptor 3.
Y Wang, M K Spatz, K Kannan, H Hayk, A Avivi, M Gorivodsky, M Pines, A Yayon, P Lonai, D Givol. Proc Natl Acad Sci U S A 1999
166
14

Thanatophoric dysplasia (types I and II) caused by distinct mutations in fibroblast growth factor receptor 3.
P L Tavormina, R Shiang, L M Thompson, Y Z Zhu, D J Wilkin, R S Lachman, W R Wilcox, D L Rimoin, D H Cohn, J J Wasmuth. Nat Genet 1995
476
14

Constitutive activation of MEK1 in chondrocytes causes Stat1-independent achondroplasia-like dwarfism and rescues the Fgfr3-deficient mouse phenotype.
Shunichi Murakami, Gener Balmes, Sandra McKinney, Zhaoping Zhang, David Givol, Benoit de Crombrugghe. Genes Dev 2004
198
13

Overexpression of CNP in chondrocytes rescues achondroplasia through a MAPK-dependent pathway.
Akihiro Yasoda, Yasato Komatsu, Hideki Chusho, Takashi Miyazawa, Ami Ozasa, Masako Miura, Tatsuya Kurihara, Tomohiro Rogi, Shoji Tanaka, Michio Suda,[...]. Nat Med 2004
262
13

Evaluation of the therapeutic potential of a CNP analog in a Fgfr3 mouse model recapitulating achondroplasia.
Florence Lorget, Nabil Kaci, Jeff Peng, Catherine Benoist-Lasselin, Emilie Mugniery, Todd Oppeneer, Dan J Wendt, Sean M Bell, Sherry Bullens, Stuart Bunting,[...]. Am J Hum Genet 2012
101
13


Cellular signaling by fibroblast growth factor receptors.
V P Eswarakumar, I Lax, J Schlessinger. Cytokine Growth Factor Rev 2005
12

FGF signaling inhibits chondrocyte proliferation and regulates bone development through the STAT-1 pathway.
M Sahni, D C Ambrosetti, A Mansukhani, R Gertner, D Levy, C Basilico. Genes Dev 1999
279
11

FGF18 is required for normal cell proliferation and differentiation during osteogenesis and chondrogenesis.
Norihiko Ohbayashi, Masaki Shibayama, Yoko Kurotaki, Mayumi Imanishi, Toshihiko Fujimori, Nobuyuki Itoh, Shinji Takada. Genes Dev 2002
326
11

Coordination of chondrogenesis and osteogenesis by fibroblast growth factor 18.
Zhonghao Liu, Jingsong Xu, Jennifer S Colvin, David M Ornitz. Genes Dev 2002
337
11


Exploring mechanisms of FGF signalling through the lens of structural biology.
Regina Goetz, Moosa Mohammadi. Nat Rev Mol Cell Biol 2013
307
11

A novel homozygous mutation in FGFR3 causes tall stature, severe lateral tibial deviation, scoliosis, hearing impairment, camptodactyly, and arachnodactyly.
Periklis Makrythanasis, Samia Temtamy, Mona S Aglan, Ghada A Otaify, Hanan Hamamy, Stylianos E Antonarakis. Hum Mutat 2014
36
30

Unique expression pattern of the FGF receptor 3 gene during mouse organogenesis.
K Peters, D Ornitz, S Werner, L Williams. Dev Biol 1993
409
11




Interaction of FGF, Ihh/Pthlh, and BMP signaling integrates chondrocyte proliferation and hypertrophic differentiation.
Eleonora Minina, Conny Kreschel, Michael C Naski, David M Ornitz, Andrea Vortkamp. Dev Cell 2002
310
10


Defective bone mineralization and osteopenia in young adult FGFR3-/- mice.
Gladys Valverde-Franco, Hanlong Liu, David Davidson, Sen Chai, Hector Valderrama-Carvajal, David Goltzman, David M Ornitz, Janet E Henderson. Hum Mol Genet 2004
96
10

Neutral endopeptidase-resistant C-type natriuretic peptide variant represents a new therapeutic approach for treatment of fibroblast growth factor receptor 3-related dwarfism.
Daniel J Wendt, Melita Dvorak-Ewell, Sherry Bullens, Florence Lorget, Sean M Bell, Jeff Peng, Sianna Castillo, Mika Aoyagi-Scharber, Charles A O'Neill, Pavel Krejci,[...]. J Pharmacol Exp Ther 2015
56
17

An activating Fgfr3 mutation affects trabecular bone formation via a paracrine mechanism during growth.
Emilie Mugniery, Romain Dacquin, Caroline Marty, Catherine Benoist-Lasselin, Marie-Christine de Vernejoul, Pierre Jurdic, Arnold Munnich, Valérie Geoffroy, Laurence Legeai-Mallet. Hum Mol Genet 2012
34
29


Conditional inactivation of FGF receptor 2 reveals an essential role for FGF signaling in the regulation of osteoblast function and bone growth.
Kai Yu, Jingsong Xu, Zhonghao Liu, Drazen Sosic, Jiansu Shao, Eric N Olson, Dwight A Towler, David M Ornitz. Development 2003
452
10


Spatio-temporal expression of FGFR 1, 2 and 3 genes during human embryo-fetal ossification.
A L Delezoide, C Benoist-Lasselin, L Legeai-Mallet, M Le Merrer, A Munnich, M Vekemans, J Bonaventure. Mech Dev 1998
136
9


Postnatal soluble FGFR3 therapy rescues achondroplasia symptoms and restores bone growth in mice.
Stéphanie Garcia, Béatrice Dirat, Thomas Tognacci, Nathalie Rochet, Xavier Mouska, Stéphanie Bonnafous, Stéphanie Patouraux, Albert Tran, Philippe Gual, Yannick Le Marchand-Brustel,[...]. Sci Transl Med 2013
55
16

Hypertrophic chondrocytes can become osteoblasts and osteocytes in endochondral bone formation.
Liu Yang, Kwok Yeung Tsang, Hoi Ching Tang, Danny Chan, Kathryn S E Cheah. Proc Natl Acad Sci U S A 2014
350
9

Intermittent PTH (1-34) injection rescues the retarded skeletal development and postnatal lethality of mice mimicking human achondroplasia and thanatophoric dysplasia.
Yangli Xie, Nan Su, Min Jin, Huabing Qi, Junbao Yang, Can Li, Xiaolan Du, Fengtao Luo, Bo Chen, Yue Shen,[...]. Hum Mol Genet 2012
32
28


Statin treatment rescues FGFR3 skeletal dysplasia phenotypes.
Akihiro Yamashita, Miho Morioka, Hiromi Kishi, Takeshi Kimura, Yasuhito Yahara, Minoru Okada, Kaori Fujita, Hideaki Sawai, Shiro Ikegawa, Noriyuki Tsumaki. Nature 2014
124
9

Regulation of rate of cartilage differentiation by Indian hedgehog and PTH-related protein.
A Vortkamp, K Lee, B Lanske, G V Segre, H M Kronenberg, C J Tabin. Science 1996
9

Molecular mechanisms of fibroblast growth factor signaling in physiology and pathology.
Artur A Belov, Moosa Mohammadi. Cold Spring Harb Perspect Biol 2013
134
9


Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.