L S Dove, L C Abbott, W H Griffith. J Neurosci 1998
Times Cited: 109
Times Cited: 109
Times Cited
Times Co-cited
Similarity
Single tottering mutations responsible for the neuropathic phenotype of the P-type calcium channel.
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Altered calcium channel currents in Purkinje cells of the neurological mutant mouse leaner.
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Absence epilepsy in tottering mutant mice is associated with calcium channel defects.
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Familial hemiplegic migraine and episodic ataxia type-2 are caused by mutations in the Ca2+ channel gene CACNL1A4.
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Autosomal dominant cerebellar ataxia (SCA6) associated with small polyglutamine expansions in the alpha 1A-voltage-dependent calcium channel.
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Ablation of P/Q-type Ca(2+) channel currents, altered synaptic transmission, and progressive ataxia in mice lacking the alpha(1A)-subunit.
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Reduced voltage sensitivity of activation of P/Q-type Ca2+ channels is associated with the ataxic mouse mutation rolling Nagoya (tg(rol)).
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Cerebellar cell degeneration in the leaner mutant mouse.
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Mutations in the Cacnl1a4 calcium channel gene are associated with seizures, cerebellar degeneration, and ataxia in tottering and leaner mutant mice.
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Functional consequences of mutations in the human alpha1A calcium channel subunit linked to familial hemiplegic migraine.
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Rocker is a new variant of the voltage-dependent calcium channel gene Cacna1a.
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Dystonia and cerebellar atrophy in Cacna1a null mice lacking P/Q calcium channel activity.
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Familial hemiplegic migraine mutations change alpha1A Ca2+ channel kinetics.
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L-type calcium channels contribute to the tottering mouse dystonic episodes.
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Presynaptic Ca(2+) influx at a mouse central synapse with Ca(2+) channel subunit mutations.
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Human epilepsy associated with dysfunction of the brain P/Q-type calcium channel.
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Inherited epilepsy: spike-wave and focal motor seizures in the mutant mouse tottering.
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Primary structure and functional expression from complementary DNA of a brain calcium channel.
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An ultrastructural study of granule cell/Purkinje cell synapses in tottering (tg/tg), leaner (tg(la)/tg(la)) and compound heterozygous tottering/leaner (tg/tg(la)) mice.
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Splicing of alpha 1A subunit gene generates phenotypic variants of P- and Q-type calcium channels.
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Purkinje cell loss from alternating sagittal zones in the cerebellum of leaner mutant mice.
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Impaired neurotransmitter release and elevated threshold for cortical spreading depression in mice with mutations in the alpha1A subunit of P/Q type calcium channels.
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Bidirectional alterations in cerebellar synaptic transmission of tottering and rolling Ca2+ channel mutant mice.
Kaori Matsushita, Minoru Wakamori, Im Joo Rhyu, Tatsuo Arii, Sen-Ichi Oda, Yasuo Mori, Keiji Imoto. J Neurosci 2002
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Calcium control of transmitter release at a cerebellar synapse.
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Excitatory but not inhibitory synaptic transmission is reduced in lethargic (Cacnb4(lh)) and tottering (Cacna1atg) mouse thalami.
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The mouse stargazer gene encodes a neuronal Ca2+-channel gamma subunit.
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Tottering and leaner mutations perturb transient developmental expression of tyrosine hydroxylase in embryologically distinct Purkinje cells.
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Tottering--a neuromusclar mutation in the mouse. And its linkage with oligosyndacylism.
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Expression of calcium channel alpha1A mRNA and protein in the leaner mouse (tgla/tgla) cerebellum.
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Altered calcium homeostasis in cerebellar Purkinje cells of leaner mutant mice.
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Calcium channels and channelopathies of the central nervous system.
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Nomenclature of voltage-gated calcium channels.
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Roles of N-type and Q-type Ca2+ channels in supporting hippocampal synaptic transmission.
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Three new familial hemiplegic migraine mutants affect P/Q-type Ca(2+) channel kinetics.
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Decreases in the precision of Purkinje cell pacemaking cause cerebellar dysfunction and ataxia.
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Pharmacological dissection of multiple types of Ca2+ channel currents in rat cerebellar granule neurons.
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Expression of tyrosine hydroxylase in cerebellar Purkinje neurons of the mutant tottering and leaner mouse.
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Complete loss of P/Q calcium channel activity caused by a CACNA1A missense mutation carried by patients with episodic ataxia type 2.
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A Cacna1a knockin migraine mouse model with increased susceptibility to cortical spreading depression.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.