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Times Cited: 173
Times Cited: 173
Times Cited
Times Co-cited
Similarity
Absence epilepsy in tottering mutant mice is associated with calcium channel defects.
C F Fletcher, C M Lutz, T N O'Sullivan, J D Shaughnessy, R Hawkes, W N Frankel, N G Copeland, N A Jenkins. Cell 1996
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Familial hemiplegic migraine and episodic ataxia type-2 are caused by mutations in the Ca2+ channel gene CACNL1A4.
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Autosomal dominant cerebellar ataxia (SCA6) associated with small polyglutamine expansions in the alpha 1A-voltage-dependent calcium channel.
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Ablation of P/Q-type Ca(2+) channel currents, altered synaptic transmission, and progressive ataxia in mice lacking the alpha(1A)-subunit.
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Whole-cell and single-channel analysis of P-type calcium currents in cerebellar Purkinje cells of leaner mutant mice.
L S Dove, L C Abbott, W H Griffith. J Neurosci 1998
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Altered calcium channel currents in Purkinje cells of the neurological mutant mouse leaner.
N M Lorenzon, C M Lutz, W N Frankel, K G Beam. J Neurosci 1998
N M Lorenzon, C M Lutz, W N Frankel, K G Beam. J Neurosci 1998
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Reduced voltage sensitivity of activation of P/Q-type Ca2+ channels is associated with the ataxic mouse mutation rolling Nagoya (tg(rol)).
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Human epilepsy associated with dysfunction of the brain P/Q-type calcium channel.
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Rocker is a new variant of the voltage-dependent calcium channel gene Cacna1a.
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Dystonia and cerebellar atrophy in Cacna1a null mice lacking P/Q calcium channel activity.
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Immunochemical identification and subcellular distribution of the alpha 1A subunits of brain calcium channels.
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Inherited epilepsy: spike-wave and focal motor seizures in the mutant mouse tottering.
J L Noebels, R L Sidman. Science 1979
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A Cacna1a knockin migraine mouse model with increased susceptibility to cortical spreading depression.
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22
Dysfunction of the brain calcium channel CaV2.1 in absence epilepsy and episodic ataxia.
Paola Imbrici, Stephen L Jaffe, Louise H Eunson, Nicholas P Davies, Colin Herd, Robert Robertson, Dimitri M Kullmann, Michael G Hanna. Brain 2004
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Presynaptic Ca(2+) influx at a mouse central synapse with Ca(2+) channel subunit mutations.
J Qian, J L Noebels. J Neurosci 2000
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Bidirectional alterations in cerebellar synaptic transmission of tottering and rolling Ca2+ channel mutant mice.
Kaori Matsushita, Minoru Wakamori, Im Joo Rhyu, Tatsuo Arii, Sen-Ichi Oda, Yasuo Mori, Keiji Imoto. J Neurosci 2002
Kaori Matsushita, Minoru Wakamori, Im Joo Rhyu, Tatsuo Arii, Sen-Ichi Oda, Yasuo Mori, Keiji Imoto. J Neurosci 2002
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Primary structure and functional expression from complementary DNA of a brain calcium channel.
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Calcium channels and channelopathies of the central nervous system.
Daniela Pietrobon. Mol Neurobiol 2002
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Mutations in the Cacnl1a4 calcium channel gene are associated with seizures, cerebellar degeneration, and ataxia in tottering and leaner mutant mice.
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Decreases in the precision of Purkinje cell pacemaking cause cerebellar dysfunction and ataxia.
Joy T Walter, Karina Alviña, Mary D Womack, Carolyn Chevez, Kamran Khodakhah. Nat Neurosci 2006
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Tottering--a neuromusclar mutation in the mouse. And its linkage with oligosyndacylism.
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Cerebellar cell degeneration in the leaner mutant mouse.
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Increased noise level of purkinje cell activities minimizes impact of their modulation during sensorimotor control.
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Splicing of alpha 1A subunit gene generates phenotypic variants of P- and Q-type calcium channels.
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Tottering mouse motor dysfunction is abolished on the Purkinje cell degeneration (pcd) mutant background.
D B Campbell, J B North, E J Hess. Exp Neurol 1999
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Impaired neurotransmitter release and elevated threshold for cortical spreading depression in mice with mutations in the alpha1A subunit of P/Q type calcium channels.
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L-type calcium channels contribute to the tottering mouse dystonic episodes.
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Localization and functional properties of a rat brain alpha 1A calcium channel reflect similarities to neuronal Q- and P-type channels.
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Pharmacological dissection of multiple types of Ca2+ channel currents in rat cerebellar granule neurons.
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Excitatory but not inhibitory synaptic transmission is reduced in lethargic (Cacnb4(lh)) and tottering (Cacna1atg) mouse thalami.
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Enhanced G protein-dependent modulation of excitatory synaptic transmission in the cerebellum of the Ca2+ channel-mutant mouse, tottering.
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P-type calcium channels blocked by the spider toxin omega-Aga-IVA.
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Complete loss of P/Q calcium channel activity caused by a CACNA1A missense mutation carried by patients with episodic ataxia type 2.
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Functional consequences of P/Q-type Ca2+ channel Cav2.1 missense mutations associated with episodic ataxia type 2 and progressive ataxia.
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An ultrastructural study of granule cell/Purkinje cell synapses in tottering (tg/tg), leaner (tg(la)/tg(la)) and compound heterozygous tottering/leaner (tg/tg(la)) mice.
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Roles of N-type and Q-type Ca2+ channels in supporting hippocampal synaptic transmission.
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Different types of calcium channels mediate central synaptic transmission.
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Lack of the burst firing of thalamocortical relay neurons and resistance to absence seizures in mice lacking alpha(1G) T-type Ca(2+) channels.
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Triggers of paroxysmal dyskinesia in the calcium channel mouse mutant tottering.
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Altered properties of quantal neurotransmitter release at endplates of mice lacking P/Q-type Ca2+ channels.
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Spinocerebellar ataxia type 6 mutation alters P-type calcium channel function.
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Abnormal transmitter release at neuromuscular junctions of mice carrying the tottering alpha(1A) Ca(2+) channel mutation.
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Ducky mouse phenotype of epilepsy and ataxia is associated with mutations in the Cacna2d2 gene and decreased calcium channel current in cerebellar Purkinje cells.
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P/Q-type calcium channels mediate the activity-dependent feedback of syntaxin-1A.
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Familial hemiplegic migraine mutations increase Ca(2+) influx through single human CaV2.1 channels and decrease maximal CaV2.1 current density in neurons.
Angelita Tottene, Tommaso Fellin, Stefano Pagnutti, Siro Luvisetto, Joerg Striessnig, Colin Fletcher, Daniela Pietrobon. Proc Natl Acad Sci U S A 2002
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.