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List of shared articles
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Recombinant pro-CTSD (cathepsin D) enhances SNCA/α-Synuclein degradation in α-Synucleinopathy models.
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Targeted attenuation of elevated histone marks at SNCA alleviates α-synuclein in Parkinson's disease.
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α-synuclein pathogenesis in hiPSC models of Parkinson's disease.
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Midbrain organoids with an SNCA gene triplication model key features of synucleinopathy.
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